Cell Signaling Technology

ID1 (F2M1J) Rabbit Monoclonal Antibody (IF/Flow Formulated)

#25344 · immunofluorescence · $161,376

40
Citing papers
1.6k
Combined citations
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Research areas

Cancer-related gene regulation5RNA modifications and cancer5RNA Research and Splicing4Curcumin's Biomedical Applications4Epigenetics and DNA Methylation4Neuropeptides and Animal Physiology3Retinoids in leukemia and cellular processes3Cancer Cells and Metastasis2

Papers citing this product

1
Betulinic acid inhibits colon cancer cell and tumor growth and induces proteasome-dependent and -independent downregulation of specificity proteins (Sp) transcription factors
BMC Cancer·2011·162 cit.·DOI
2
miR-7 and miR-153 protect neurons against MPP+-induced cell death via upregulation of mTOR pathway
Frontiers in Cellular Neuroscience·2014·153 cit.·DOI
3
Aspirin Inhibits Colon Cancer Cell and Tumor Growth and Downregulates Specificity Protein (Sp) Transcription Factors
PLoS ONE·2012·127 cit.·DOI
4
Combination of quercetin and hyperoside has anticancer effects on renal cancer cells through inhibition of oncogenic microRNA-27a
Oncology Reports·2013·115 cit.·DOI
5
Improved modeling of human AD with an automated culturing platform for iPSC neurons, astrocytes and microglia
Nature Communications·2021·109 cit.·DOI
6
Curcumin reverses the epithelial-mesenchymal transition of pancreatic cancer cells by inhibiting the Hedgehog signaling pathway
Oncology Reports·2013·109 cit.·DOI
7
RNA Binding Protein CUGBP2/CELF2 Mediates Curcumin-Induced Mitotic Catastrophe of Pancreatic Cancer Cells
PLoS ONE·2011·83 cit.·DOI
8
Biochemical Control of CARM1 Enzymatic Activity by Phosphorylation
Journal of Biological Chemistry·2009·77 cit.·DOI
9
Cyclooxygenase-2 contributes to oxidopamine-mediated neuronal inflammation and injury via the prostaglandin E2 receptor EP2 subtype
Scientific Reports·2017·64 cit.·DOI
10
High Concentrations of L-Ascorbic Acid Specifically Inhibit the Growth of Human Leukemic Cells via Downregulation of HIF-1α Transcription
PLoS ONE·2013·53 cit.·DOI

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