Cell Signaling Technology

IL-18 (D2F3B) Rabbit Monoclonal Antibody (BSA and Azide Free)

#46202 · $865

70
Evidence Score · Well-Established
Used by 148 independent labs across 166 papers. How we score this →
16
Citing papers
1.4k
Combined citations
Supplier page Find this product

Research areas

Metabolism, Diabetes, and Cancer3Immune Cell Function and Interaction2Click Chemistry and Applications2Kruppel-like factors research2Cancer-related gene regulation1Cancer, Stress, Anesthesia, and Immune Response1Cancer Immunotherapy and Biomarkers1Cancer Mechanisms and Therapy1

Papers citing this product

1
The role of AMP-activated protein kinase in the action of ethanol in the liver
Gastroenterology·2004·455 cit.·DOI
2
Chromium Activates Glucose Transporter 4 Trafficking and Enhances Insulin-Stimulated Glucose Transport in 3T3-L1 Adipocytes via a Cholesterol-Dependent Mechanism
Molecular Endocrinology·2005·184 cit.·DOI
3
Phosphoinositide-3-kinase and mitogen activated protein kinase signaling pathways mediate acute NGF sensitization of TRPV1
Molecular and Cellular Neuroscience·2007·161 cit.·DOI
4
Mechanisms Regulating the Constitutive Activation of the Extracellular Signal-Regulated Kinase (ERK) Signaling Pathway in Ovarian Cancer and the Effect of Ribonucleic Acid Interference for ERK1/2 on Cancer Cell Proliferation
Molecular Endocrinology·2004·135 cit.·DOI
5
Bacterial sepsis triggers an antiviral response that causes translation shutdown
Journal of Clinical Investigation·2018·62 cit.·DOI
6
Imbalanced mitochondrial function provokes heterotaxy via aberrant ciliogenesis
Journal of Clinical Investigation·2019·60 cit.·DOI
7
ST2 as checkpoint target for colorectal cancer immunotherapy
JCI Insight·2020·52 cit.·DOI
8
Loss of miR-29a/b1 promotes inflammation and fibrosis in acute pancreatitis
JCI Insight·2021·47 cit.·DOI
9
Glucocorticoid-Induced Suppression of β-Cell Proliferation Is Mediated by Mig6
Endocrinology·2013·41 cit.·DOI
10
STAT4 is expressed in neutrophils and promotes antimicrobial immunity
JCI Insight·2021·39 cit.·DOI

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