Cell Signaling Technology

USP10 Antibody

#5553 · $414

100
Citing papers
8k
Combined citations
Supplier page Find this product

Research areas

MicroRNA in disease regulation15Extracellular vesicles in disease13Circular RNAs in diseases11Cancer-related molecular mechanisms research7Immune cells in cancer7RNA modifications and cancer7Cancer, Hypoxia, and Metabolism6Cancer Cells and Metastasis5

Papers citing this product

1
Toll-like Receptors Induce a Phagocytic Gene Program through p38
The Journal of Experimental Medicine·2003·428 cit.·DOI
2
Differential Requirement for TANK-binding Kinase-1 in Type I Interferon Responses to Toll-like Receptor Activation and Viral Infection
The Journal of Experimental Medicine·2004·400 cit.·DOI
3
KIBRA controls exosome secretion via inhibiting the proteasomal degradation of Rab27a
Nature Communications·2019·258 cit.·DOI
4
Plaque associated microglia hyper-secrete extracellular vesicles and accelerate tau propagation in a humanized APP mouse model
Molecular Neurodegeneration·2021·230 cit.·DOI
5
RETRACTED ARTICLE: Exosomal microRNA-32-5p induces multidrug resistance in hepatocellular carcinoma via the PI3K/Akt pathway
Journal of Experimental & Clinical Cancer Research·2018·222 cit.·DOI
6
TNFα Is Required for Late BRB Breakdown in Diabetic Retinopathy, and Its Inhibition Prevents Leukostasis and Protects Vessels and Neurons from Apoptosis
Investigative Ophthalmology & Visual Science·2011·216 cit.·DOI
7
Angiopoietin-1 Requires p190 RhoGAP to Protect against Vascular Leakage in Vivo
Journal of Biological Chemistry·2007·180 cit.·DOI
8
WNT/β-catenin-suppressed FTO expression increases m6A of c-Myc mRNA to promote tumor cell glycolysis and tumorigenesis
Cell Death and Disease·2021·170 cit.·DOI
9
Human-Specific ARHGAP11B Acts in Mitochondria to Expand Neocortical Progenitors by Glutaminolysis
Neuron·2019·151 cit.·DOI
10
Respiratory virus–induced EGFR activation suppresses IRF1-dependent interferon λ and antiviral defense in airway epithelium
The Journal of Experimental Medicine·2013·142 cit.·DOI

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