Cell Signaling Technology

TDRD3 (D3O2G) Rabbit Monoclonal Antibody

#5942 · western blot · $161,351

100

Citing papers

10k

Combined citations

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Research areas

Autophagy in Disease and Therapy17 Wnt/β-catenin signaling in development and cancer15 Cancer-related gene regulation8 Cancer Cells and Metastasis6 Neurogenesis and neuroplasticity mechanisms5 Sirtuins and Resveratrol in Medicine5 Endoplasmic Reticulum Stress and Disease5 Epigenetics and DNA Methylation5

Papers citing this product

1

Dysregulation of the Wnt pathway inhibits timely myelination and remyelination in the mammalian CNS

Genes & Development·2009·632 cit.·DOI

2

Nuclear condensates of the Polycomb protein chromobox 2 (CBX2) assemble through phase separation

Journal of Biological Chemistry·2018·359 cit.·DOI

3

The ER stress factor XBP1s prevents amyloid-β neurotoxicity

Human Molecular Genetics·2011·282 cit.·DOI

4

BDNF Promotes Differentiation and Maturation of Adult-born Neurons through GABAergic Transmission

Journal of Neuroscience·2012·282 cit.·DOI

5

Histone H2A Mono-Ubiquitination Is a Crucial Step to Mediate PRC1-Dependent Repression of Developmental Genes to Maintain ES Cell Identity

PLoS Genetics·2012·254 cit.·DOI

6

EGFR Tyrosine Kinase Inhibitors Activate Autophagy as a Cytoprotective Response in Human Lung Cancer Cells

PLoS ONE·2011·252 cit.·DOI

7

Multiple TLRs Are Expressed in Human Cholangiocytes and Mediate Host Epithelial Defense Responses to <i>Cryptosporidium parvum</i> via Activation of NF-κB

The Journal of Immunology·2005·238 cit.·DOI

8

Signaling through BMP receptors promotes respiratory identity in the foregut via repression of <i>Sox2</i>

Development·2011·209 cit.·DOI

9

Canonical Wnt signaling functions in second heart field to promote right ventricular growth

Proceedings of the National Academy of Sciences·2007·193 cit.·DOI

10

Resveratrol-induced autophagy and apoptosis in cisplatin-resistant human oral cancer CAR cells: A key role of AMPK and Akt/mTOR signaling

International Journal of Oncology·2017·191 cit.·DOI

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