Cell Signaling Technology

ThPOK (D9V5T) Rabbit Monoclonal Antibody (BSA and Azide Free)

#20560 · $865

100
Citing papers
2.5k
Combined citations
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Research areas

Osteoarthritis Treatment and Mechanisms18Autophagy in Disease and Therapy12Cell death mechanisms and regulation10interferon and immune responses7Neuroinflammation and Neurodegeneration Mechanisms6NF-κB Signaling Pathways6Cancer-related molecular mechanisms research6Epigenetics and DNA Methylation5

Papers citing this product

1
MTORC1 coordinates the autophagy and apoptosis signaling in articular chondrocytes in osteoarthritic temporomandibular joint
Autophagy·2019·291 cit.·DOI
2
The role of microglial P2X7: modulation of cell death and cytokine release
Journal of Neuroinflammation·2017·200 cit.·DOI
3
Induction of lysosomal and mitochondrial biogenesis by AMPK phosphorylation of FNIP1
Science·2023·197 cit.·DOI
4
SGK1 inhibition induces autophagy-dependent apoptosis via the mTOR-Foxo3a pathway
British Journal of Cancer·2017·117 cit.·DOI
5
Heat shock protein 90α (HSP90α), a substrate and chaperone of DNA-PK necessary for the apoptotic response
Proceedings of the National Academy of Sciences·2012·83 cit.·DOI
6
<p>Overexpression of SOX9 alleviates the progression of human osteoarthritis in vitro and in vivo</p>
Drug Design Development and Therapy·2019·66 cit.·DOI
7
Protease-activated receptor 2 (PAR-2) antagonist AZ3451 as a novel therapeutic agent for osteoarthritis
Aging·2019·65 cit.·DOI
8
TRAP1 regulates stemness through Wnt/β-catenin pathway in human colorectal carcinoma
Cell Death and Differentiation·2016·59 cit.·DOI
9
Near-infrared light reduces β-amyloid-stimulated microglial toxicity and enhances survival of neurons: mechanisms of light therapy for Alzheimer’s disease
Alzheimer s Research & Therapy·2022·56 cit.·DOI
10
IL-1β receptor antagonist (IL-1Ra) combined with autophagy inducer (TAT-Beclin1) is an effective alternative for attenuating extracellular matrix degradation in rat and human osteoarthritis chondrocytes
Arthritis Research & Therapy·2019·56 cit.·DOI

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