Cell Signaling Technology

MLKL (D2I6N) Rabbit Monoclonal Antibody

#14993 · $173,351

68
Evidence Score · Well-Established
Used by 163 independent labs across 184 papers. How we score this →
100
Citing papers
3.5k
Combined citations
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Research areas

Cell death mechanisms and regulation19interferon and immune responses9Inflammasome and immune disorders8Ubiquitin and proteasome pathways7Mitochondrial Function and Pathology6Autophagy in Disease and Therapy5Immune Cell Function and Interaction5Immune cells in cancer5

Papers citing this product

1
Forkhead transcription factor FOXO1 (FKHR)-dependent induction of PDK4 gene expression in skeletal muscle during energy deprivation
Biochemical Journal·2003·326 cit.·DOI
2
Dynamic regulation of PGC‐1α localization and turnover implicates mitochondrial adaptation in calorie restriction and the stress response
Aging Cell·2007·282 cit.·DOI
3
Myeloid-derived suppressor cells endow stem-like qualities to multiple myeloma cells by inducing piRNA-823 expression and DNMT3B activation
Molecular Cancer·2019·143 cit.·DOI
4
SARS-CoV-2 Z-RNA activates the ZBP1-RIPK3 pathway to promote virus-induced inflammatory responses
Cell Research·2023·100 cit.·DOI
5
Reduced Activity of AMP-Activated Protein Kinase Protects against Genetic Models of Motor Neuron Disease
Journal of Neuroscience·2012·98 cit.·DOI
6
A critical role of IL-17 in modulating the B-cell response during H5N1 influenza virus infection
Cellular and Molecular Immunology·2011·97 cit.·DOI
7
The SIAH2-NRF1 axis spatially regulates tumor microenvironment remodeling for tumor progression
Nature Communications·2019·90 cit.·DOI
8
Exercise training reverses cardiac aging phenotypes associated with heart failure with preserved ejection fraction in male mice
Aging Cell·2020·80 cit.·DOI
9
Caloric Restriction Shortens Lifespan through an Increase in Lipid Peroxidation, Inflammation and Apoptosis in the G93A Mouse, an Animal Model of ALS
PLoS ONE·2010·73 cit.·DOI
10
BAFF Promotes Th17 Cells and Aggravates Experimental Autoimmune Encephalomyelitis
PLoS ONE·2011·71 cit.·DOI

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