Cell Signaling Technology

MSK2 (D41A4) Rabbit Monoclonal Antibody

#3679 · $175

100
Citing papers
12k
Combined citations
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Research areas

Neuroscience and Neuropharmacology Research11PI3K/AKT/mTOR signaling in cancer7Cancer-related Molecular Pathways5Cell death mechanisms and regulation5Acute Myeloid Leukemia Research5Adipose Tissue and Metabolism5interferon and immune responses5Retinal Development and Disorders5

Papers citing this product

1
Activation of the Phosphoinositide 3-Kinase-Akt-Mammalian Target of Rapamycin Signaling Pathway Is Required for Metabotropic Glutamate Receptor-Dependent Long-Term Depression
Journal of Neuroscience·2004·516 cit.·DOI
2
Phosphatidylinositol 3-Kinase Activates ERK in Primary Sensory Neurons and Mediates Inflammatory Heat Hyperalgesia through TRPV1 Sensitization
Journal of Neuroscience·2004·415 cit.·DOI
3
Site-specific analysis of protein S-acylation by resin-assisted capture
Journal of Lipid Research·2010·348 cit.·DOI
4
SCF-mediated mast cell infiltration and activation exacerbate the inflammation and immunosuppression in tumor microenvironment
Blood·2008·344 cit.·DOI
5
Role of glutamine and interlinked asparagine metabolism in vessel formation
The EMBO Journal·2017·330 cit.·DOI
6
Internal tandem duplication of FLT3 (FLT3/ITD) induces increased ROS production, DNA damage, and misrepair: implications for poor prognosis in AML
Blood·2008·305 cit.·DOI
7
Amino Acid-Induced Translation of TOP mRNAs Is Fully Dependent on Phosphatidylinositol 3-Kinase-Mediated Signaling, Is Partially Inhibited by Rapamycin, and Is Independent of S6K1 and rpS6 Phosphorylation
Molecular and Cellular Biology·2001·298 cit.·DOI
8
Mismatch Repair Genes Mlh1 and Mlh3 Modify CAG Instability in Huntington's Disease Mice: Genome-Wide and Candidate Approaches
PLoS Genetics·2013·256 cit.·DOI
9
Regulation of Eukaryotic Initiation Factor 4E by Converging Signaling Pathways during Metabotropic Glutamate Receptor-Dependent Long-Term Depression
Journal of Neuroscience·2006·244 cit.·DOI
10
Brain-Derived Neurotrophic Factor-Dependent Synaptic Plasticity Is Suppressed by Interleukin-1β via p38 Mitogen-Activated Protein Kinase
Journal of Neuroscience·2012·242 cit.·DOI