Cell Signaling Technology

Phospho-SQSTM1/p62 (Ser349) (E7M1A) Rabbit Monoclonal Antibody (Alexa Fluor® 488 Conjugate)

#12074 · $493

100
Citing papers
3.4k
Combined citations
Supplier page Find this product

Research areas

Muscle Physiology and Disorders5Autophagy in Disease and Therapy5Extracellular vesicles in disease5Melanoma and MAPK Pathways5Cancer, Lipids, and Metabolism5Cytokine Signaling Pathways and Interactions4GDF15 and Related Biomarkers4Cancer-related Molecular Pathways4

Papers citing this product

1
Cholesterol targeting alters lipid raft composition and cell survival in prostate cancer cells and xenografts
Journal of Clinical Investigation·2005·481 cit.·DOI
2
Autophagy Regulates Endoplasmic Reticulum Homeostasis and Calcium Mobilization in T Lymphocytes
The Journal of Immunology·2010·216 cit.·DOI
3
Temporal Regulation of Intracellular Organelle Homeostasis in T Lymphocytes by Autophagy
The Journal of Immunology·2011·194 cit.·DOI
4
Antioxidative Effects of Carrot-Derived Nanovesicles in Cardiomyoblast and Neuroblastoma Cells
Pharmaceutics·2021·165 cit.·DOI
5
A Functional Nuclear Epidermal Growth Factor Receptor, Src and Stat3 Heteromeric Complex in Pancreatic Cancer Cells
PLoS ONE·2011·162 cit.·DOI
6
Interleukin-6–dependent gene expression profiles in multiple myeloma INA-6 cells reveal a Bcl-2 family–independent survival pathway closely associated with Stat3 activation
Blood·2003·152 cit.·DOI
7
Local Phosphatidylinositol 3,4,5-Trisphosphate Accumulation Recruits Vav2 and Vav3 to Activate Rac1/Cdc42 and Initiate Neurite Outgrowth in Nerve Growth Factor-stimulated PC12 Cells
Molecular Biology of the Cell·2005·144 cit.·DOI
8
Essential Role of Caveolae in Interleukin-6- and Insulin-like Growth Factor I-triggered Akt-1-mediated Survival of Multiple Myeloma Cells
Journal of Biological Chemistry·2003·144 cit.·DOI
9
Analysis of Stat3 (signal transducer and activator of transcription 3) dimerization by fluorescence resonance energy transfer in living cells
Biochemical Journal·2004·108 cit.·DOI
10
DYRK1A controls the transition from proliferation to quiescence during lymphoid development by destabilizing Cyclin D3
The Journal of Experimental Medicine·2015·84 cit.·DOI

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