Cell Signaling Technology

LKB1 (27D10) Rabbit Monoclonal Antibody

#3050 · western blot · $161,351

66
Evidence Score · Well-Established
Used by 304 independent labs across 337 papers. How we score this →
100
Citing papers
19.2k
Combined citations
Supplier page Find this product

Research areas

Autophagy in Disease and Therapy13PI3K/AKT/mTOR signaling in cancer7Chronic Lymphocytic Leukemia Research7Wnt/β-catenin signaling in development and cancer6Endoplasmic Reticulum Stress and Disease6Hippo pathway signaling and YAP/TAZ5Metabolism, Diabetes, and Cancer5Immune Response and Inflammation4

Papers citing this product

1
Proteasome inhibitors induce a terminal unfolded protein response in multiple myeloma cells
Blood·2006·1.1k cit.·DOI
2
Targeting photodynamic and photothermal therapy to the endoplasmic reticulum enhances immunogenic cancer cell death
Nature Communications·2019·1.1k cit.·DOI
3
Analytical Modeling in Applied Electromagnetics
Medical Entomology and Zoology·2003·940 cit.·DOI
4
Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure
Journal of Clinical Investigation·2005·929 cit.·DOI
5
CAL-101, a p110δ selective phosphatidylinositol-3-kinase inhibitor for the treatment of B-cell malignancies, inhibits PI3K signaling and cellular viability
Blood·2010·717 cit.·DOI
6
Class I histone deacetylases (HDAC1–3) are histone lysine delactylases
Science Advances·2022·660 cit.·DOI
7
FYCO1 is a Rab7 effector that binds to LC3 and PI3P to mediate microtubule plus end–directed vesicle transport
The Journal of Cell Biology·2010·639 cit.·DOI
8
Single-cell transcriptomics reveals regulators underlying immune cell diversity and immune subtypes associated with prognosis in nasopharyngeal carcinoma
Cell Research·2020·483 cit.·DOI
9
Vascular Endothelium As a Contributor of Plasma Sphingosine 1-Phosphate
Circulation Research·2008·474 cit.·DOI
10
RETRACTED: Tanshinone I attenuates the malignant biological properties of ovarian cancer by inducing apoptosis and autophagy via the inactivation of PI3K/AKT/mTOR pathway
Cell Proliferation·2019·351 cit.·DOI

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