Cell Signaling Technology

SignalSlide® Mouse PD-L1 IHC Controls

#41965 · $196

100
Citing papers
3.4k
Combined citations
Supplier page Find this product

Research areas

CRISPR and Genetic Engineering10Autophagy in Disease and Therapy10Ubiquitin and proteasome pathways8PI3K/AKT/mTOR signaling in cancer7Cancer, Hypoxia, and Metabolism6Cancer-related Molecular Pathways6Genetic Neurodegenerative Diseases5Cell death mechanisms and regulation5

Papers citing this product

1
Dimethylfumarate inhibits microglial and astrocytic inflammation by suppressing the synthesis of nitric oxide, IL-1β, TNF-α and IL-6 in an in-vitro model of brain inflammation
Journal of Neuroinflammation·2010·348 cit.·DOI
2
Fin56-induced ferroptosis is supported by autophagy-mediated GPX4 degradation and functions synergistically with mTOR inhibition to kill bladder cancer cells
Cell Death and Disease·2021·297 cit.·DOI
3
CD8+ T cells induce interferon-responsive oligodendrocytes and microglia in white matter aging
Nature Neuroscience·2022·207 cit.·DOI
4
Long-lasting pro-inflammatory suppression of microglia by LPS-preconditioning is mediated by RelB-dependent epigenetic silencing
Brain Behavior and Immunity·2015·187 cit.·DOI
5
Ghrelin-AMPK Signaling Mediates the Neuroprotective Effects of Calorie Restriction in Parkinson's Disease
Journal of Neuroscience·2016·161 cit.·DOI
6
DJ-1 is an essential downstream mediator in PINK1/parkin-dependent mitophagy
Brain·2022·140 cit.·DOI
7
IL1β Promotes Immune Suppression in the Tumor Microenvironment Independent of the Inflammasome and Gasdermin D
Cancer Immunology Research·2020·95 cit.·DOI
8
Human MC4R variants affect endocytosis, trafficking and dimerization revealing multiple cellular mechanisms involved in weight regulation
Cell Reports·2021·83 cit.·DOI
9
Next-generation sequencing reveals two populations of damage-induced small RNAs at endogenous DNA double-strand breaks
Nucleic Acids Research·2018·83 cit.·DOI
10
Recombinant pro-CTSD (cathepsin D) enhances SNCA/α-Synuclein degradation in α-Synucleinopathy models
Autophagy·2022·81 cit.·DOI