Cell Signaling Technology

FAAH1 Antibody

#9179 · $317

100
Citing papers
3.8k
Combined citations
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Research areas

Wnt/β-catenin signaling in development and cancer13Cancer Cells and Metastasis8Cancer-related gene regulation7Cancer-related molecular mechanisms research6Ubiquitin and proteasome pathways6Alzheimer's disease research and treatments4Birth, Development, and Health4Ferroptosis and cancer prognosis4

Papers citing this product

1
FTH1 Inhibits Ferroptosis Through Ferritinophagy in the 6-OHDA Model of Parkinson's Disease
Neurotherapeutics·2020·425 cit.·DOI
2
Homeostatic MyD88-dependent signals cause lethal inflamMation in the absence of A20
The Journal of Experimental Medicine·2008·263 cit.·DOI
3
Wnt Isoform-Specific Interactions with Coreceptor Specify Inhibition or Potentiation of Signaling by LRP6 Antibodies
PLoS ONE·2010·204 cit.·DOI
4
Systematic Mapping of WNT-FZD Protein Interactions Reveals Functional Selectivity by Distinct WNT-FZD Pairs
Journal of Biological Chemistry·2015·186 cit.·DOI
5
Apigenin inhibits pancreatic cancer cell proliferation through G2/M cell cycle arrest
Molecular Cancer·2006·183 cit.·DOI
6
Locally organised and activated Fth1hi neutrophils aggravate inflammation of acute lung injury in an IL-10-dependent manner
Nature Communications·2022·127 cit.·DOI
7
Bispecific Designed Ankyrin Repeat Proteins (DARPins) Targeting Epidermal Growth Factor Receptor Inhibit A431 Cell Proliferation and Receptor Recycling
Journal of Biological Chemistry·2011·109 cit.·DOI
8
Ferritinophagy‐Mediated Ferroptosis Involved in Paraquat‐Induced Neurotoxicity of Dopaminergic Neurons: Implication for Neurotoxicity in PD
Oxidative Medicine and Cellular Longevity·2021·103 cit.·DOI
9
Interconnected feedback loops among ESRP1, HAS2, and CD44 regulate epithelial-mesenchymal plasticity in cancer
APL Bioengineering·2018·91 cit.·DOI
10
Suppression of Cyclin-Dependent Kinase 5 Activation by Amyloid Precursor Protein: A Novel Excitoprotective Mechanism Involving Modulation of Tau Phosphorylation
Journal of Neuroscience·2005·82 cit.·DOI

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