Cell Signaling Technology

SirT1 (D60E1) Rabbit Monoclonal Antibody

#3931 · $158,348

64

Evidence Score · Well-Established

Used by 655 independent labs across 698 papers. How we score this →

100

Citing papers

9.7k

Combined citations

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Research areas

Histone Deacetylase Inhibitors Research9 Immune Cell Function and Interaction9 Myeloproliferative Neoplasms: Diagnosis and Treatment6 Cytokine Signaling Pathways and Interactions6 Cancer-related molecular mechanisms research6 MicroRNA in disease regulation6 Epigenetics and DNA Methylation5 Cell death mechanisms and regulation4

Papers citing this product

1

An amino-bisphosphonate targets MMP-9–expressing macrophages and angiogenesis to impair cervical carcinogenesis

Journal of Clinical Investigation·2004·609 cit.·DOI

2

An amino-bisphosphonate targets MMP-9–expressing macrophages and angiogenesis to impair cervical carcinogenesis

Journal of Clinical Investigation·2004·576 cit.·DOI

3

Cytosolic pH is a second messenger for glucose and regulates the PKA pathway through V‐ATPase

The EMBO Journal·2010·293 cit.·DOI

4

A Cellular-Resolution Atlas of the Larval Zebrafish Brain

Neuron·2019·288 cit.·DOI

5

Intratumoral neutrophils and plasmacytoid dendritic cells indicate poor prognosis and are associated with pSTAT3 expression in AJCC stage I/II melanoma

Cancer·2011·280 cit.·DOI

6

A circular transcript of <i>ncx1</i> gene mediates ischemic myocardial injury by targeting miR-133a-3p

Theranostics·2018·238 cit.·DOI

7

Regulation of NMDA Receptors by Neuregulin Signaling in Prefrontal Cortex

Journal of Neuroscience·2005·216 cit.·DOI

8

TRPM2 Contributes to Inflammatory and Neuropathic Pain through the Aggravation of Pronociceptive Inflammatory Responses in Mice

Journal of Neuroscience·2012·205 cit.·DOI

9

Canonical Wnt signaling functions in second heart field to promote right ventricular growth

Proceedings of the National Academy of Sciences·2007·193 cit.·DOI

10

Glucose represses dendritic cell-induced T cell responses

Nature Communications·2017·182 cit.·DOI

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