Cell Signaling Technology

Staurosporine

#9953 · $271

100
Citing papers
4.3k
Combined citations
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Research areas

Neurogenesis and neuroplasticity mechanisms8PI3K/AKT/mTOR signaling in cancer8Neuroinflammation and Neurodegeneration Mechanisms5Cancer-related Molecular Pathways5Autophagy in Disease and Therapy5Cell death mechanisms and regulation5Metabolism, Diabetes, and Cancer5Nerve injury and regeneration5

Papers citing this product

1
Caffeine induces apoptosis by enhancement of autophagy via PI3K/Akt/mTOR/p70S6K inhibition
Autophagy·2011·419 cit.·DOI
2
Defective insulin secretion and increased susceptibility to experimental diabetes are induced by reduced Akt activity in pancreatic islet β cells
Journal of Clinical Investigation·2004·212 cit.·DOI
3
A Bacterial Acetyltransferase Destroys Plant Microtubule Networks and Blocks Secretion
PLoS Pathogens·2012·187 cit.·DOI
4
Low-temperature plasma treatment induces DNA damage leading to necrotic cell death in primary prostate epithelial cells
British Journal of Cancer·2015·173 cit.·DOI
5
c-Abl regulates YAPY357 phosphorylation to activate endothelial atherogenic responses to disturbed flow
Journal of Clinical Investigation·2019·137 cit.·DOI
6
Unraveling a Three-Step Spatiotemporal Mechanism of Triggering of Receptor-Induced Nipah Virus Fusion and Cell Entry
PLoS Pathogens·2013·133 cit.·DOI
7
Norrin Mediates Neuroprotective Effects on Retinal Ganglion Cells via Activation of the Wnt/β-Catenin Signaling Pathway and the Induction of Neuroprotective Growth Factors in Müller Cells
Journal of Neuroscience·2010·132 cit.·DOI
8
Polymorphic regulation of mitochondrial fission and fusion modifies phenotypes of microglia in neuroinflammation
Scientific Reports·2017·116 cit.·DOI
9
Remodelling of tumour microenvironment by microwave ablation potentiates immunotherapy of AXL-specific CAR T cells against non-small cell lung cancer
Nature Communications·2022·104 cit.·DOI
10
Chemoprevention and Treatment of Experimental Cowden's Disease by mTOR Inhibition with Rapamycin
Cancer Research·2008·102 cit.·DOI