Cell Signaling Technology

Progesterone Receptor B (C1A2) Rabbit Monoclonal Antibody

#3157 · $159

100
Citing papers
12.3k
Combined citations
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Research areas

Cell death mechanisms and regulation9Cytokine Signaling Pathways and Interactions8Autophagy in Disease and Therapy7Cell Adhesion Molecules Research6MicroRNA in disease regulation5Cancer-related Molecular Pathways5RNA Interference and Gene Delivery5S100 Proteins and Annexins5

Papers citing this product

1
Trop-2 is a novel target for solid cancer therapy with sacituzumab govitecan (IMMU-132), an antibody-drug conjugate (ADC)*
Oncotarget·2015·464 cit.·DOI
2
Perifosine, an oral bioactive novel alkylphospholipid, inhibits Akt and induces in vitro and in vivo cytotoxicity in human multiple myeloma cells
Blood·2006·421 cit.·DOI
3
Epigenetic Regulation of Motor Neuron Cell Death through DNA Methylation
Journal of Neuroscience·2011·360 cit.·DOI
4
Release of cytochrome c and activation of pro-caspase-9 following lysosomal photodamage involves bid cleavage
Cell Death and Differentiation·2002·321 cit.·DOI
5
Overlapping cleavage motif selectivity of caspases: implications for analysis of apoptotic pathways
Cell Death and Differentiation·2007·310 cit.·DOI
6
Glucagon-like peptide-1 inhibits adipose tissue macrophage infiltration and inflammation in an obese mouse model of diabetes
Diabetologia·2012·301 cit.·DOI
7
Nurselike cells express BAFF and APRIL, which can promote survival of chronic lymphocytic leukemia cells via a paracrine pathway distinct from that of SDF-1α
Blood·2005·300 cit.·DOI
8
Girls homozygous for an IL-2–inducible T cell kinase mutation that leads to protein deficiency develop fatal EBV-associated lymphoproliferation
Journal of Clinical Investigation·2009·289 cit.·DOI
9
Translational and Therapeutic Evaluation of RAS-GTP Inhibition by RMC-6236 in RAS-Driven Cancers
Cancer Discovery·2024·267 cit.·DOI
10
Cathepsin D mediates cytochrome c release and caspase activation in human fibroblast apoptosis induced by staurosporine
Cell Death and Differentiation·2003·231 cit.·DOI

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