Cell Signaling Technology

RSK1/RSK2/RSK3 Antibody

#9347 · $368

100
Citing papers
3.3k
Combined citations
Supplier page Find this product

Research areas

Cytokine Signaling Pathways and Interactions11interferon and immune responses8PI3K/AKT/mTOR signaling in cancer6RNA modifications and cancer6MicroRNA in disease regulation6Neuroscience and Neuropharmacology Research5Cancer Cells and Metastasis5Cancer, Hypoxia, and Metabolism5

Papers citing this product

1
Endothelin-1 Regulates Astrocyte Proliferation and Reactive Gliosis via a JNK/c-Jun Signaling Pathway
Journal of Neuroscience·2008·244 cit.·DOI
2
Mitochondrial dysfunction in some triple-negative breast cancer cell lines: role of mTOR pathway and therapeutic potential
Breast Cancer Research·2014·218 cit.·DOI
3
Pseudomonas aeruginosa Pili and Flagella Mediate Distinct Binding and Signaling Events at the Apical and Basolateral Surface of Airway Epithelium
PLoS Pathogens·2012·209 cit.·DOI
4
γ-Protocadherins regulate neuronal survival but are dispensable for circuit formation in retina
Development·2008·160 cit.·DOI
5
Endothelin-1 Activates Endothelial Cell Nitric-oxide Synthase via Heterotrimeric G-protein βγ Subunit Signaling to Protein Kinase B/Akt
Journal of Biological Chemistry·2003·144 cit.·DOI
6
ERK5 signalling rescues intestinal epithelial turnover and tumour cell proliferation upon ERK1/2 abrogation
Nature Communications·2016·105 cit.·DOI
7
Autophagy Inhibitor Chloroquine Enhanced the Cell Death Inducing Effect of the Flavonoid Luteolin in Metastatic Squamous Cell Carcinoma Cells
PLoS ONE·2012·93 cit.·DOI
8
Dietary Flavonoids Luteolin and Quercetin Inhibit Migration and Invasion of Squamous Carcinoma through Reduction of Src/Stat3/S100A7 Signaling
Antioxidants·2019·90 cit.·DOI
9
Suppression of Insulin Receptor Substrate 1 (IRS-1) Promotes Mammary Tumor Metastasis
Molecular and Cellular Biology·2006·88 cit.·DOI
10
Activating Transcription Factor 3 Deficiency Promotes Cardiac Hypertrophy, Dysfunction, and Fibrosis Induced by Pressure Overload
PLoS ONE·2011·82 cit.·DOI

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