Cell Signaling Technology

FIP200 (D10D11) Rabbit Monoclonal Antibody

#12436 · $161

100
Citing papers
3.3k
Combined citations
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Research areas

Autophagy in Disease and Therapy15Cellular transport and secretion8Cancer-related molecular mechanisms research7Cancer, Hypoxia, and Metabolism6Cell Adhesion Molecules Research6Pulmonary Hypertension Research and Treatments5Cancer-related Molecular Pathways5Ubiquitin and proteasome pathways5

Papers citing this product

1
Impaired mitophagy links mitochondrial disease to epithelial stress in methylmalonyl-CoA mutase deficiency
Nature Communications·2020·142 cit.·DOI
2
GABARAP sequesters the FLCN-FNIP tumor suppressor complex to couple autophagy with lysosomal biogenesis
Science Advances·2021·124 cit.·DOI
3
VGF and Its C-Terminal Peptide TLQP-62 Regulate Memory Formation in Hippocampus via a BDNF-TrkB-Dependent Mechanism
Journal of Neuroscience·2015·122 cit.·DOI
4
Noncanonical function of an autophagy protein prevents spontaneous Alzheimer’s disease
Science Advances·2020·115 cit.·DOI
5
Baicalin alleviates bleomycin‑induced pulmonary fibrosis and fibroblast proliferation in rats via the PI3K/AKT signaling pathway
Molecular Medicine Reports·2020·111 cit.·DOI
6
Luteolin Ameliorates Experimental Pulmonary Arterial Hypertension via Suppressing Hippo-YAP/PI3K/AKT Signaling Pathway
Frontiers in Pharmacology·2021·94 cit.·DOI
7
Role of Cyclin B1/Cdc2 Up-Regulation in the Development of Mitotic Prometaphase Arrest in Human Breast Cancer Cells Treated with Nocodazole
PLoS ONE·2011·94 cit.·DOI
8
Lysyl Oxidase Oxidizes Cell Membrane Proteins and Enhances the Chemotactic Response of Vascular Smooth Muscle Cells
Journal of Biological Chemistry·2008·82 cit.·DOI
9
Vorinostat suppresses hypoxia signaling by modulating nuclear translocation of hypoxia inducible factor 1 alpha
Oncotarget·2017·81 cit.·DOI
10
Human Fetal-Derived Enterospheres Provide Insights on Intestinal Development and a Novel Model to Study Necrotizing Enterocolitis (NEC)
Cellular and Molecular Gastroenterology and Hepatology·2018·80 cit.·DOI