Cell Signaling Technology

CD36 Antibody

#74002 · $317

10
Citing papers
306
Combined citations
Supplier page Find this product

Research areas

Nuclear Structure and Function2FOXO transcription factor regulation2Hormonal Regulation and Hypertension2Nitric Oxide and Endothelin Effects2Atherosclerosis and Cardiovascular Diseases2Diet and metabolism studies1Circular RNAs in diseases1Cancer, Hypoxia, and Metabolism1

Papers citing this product

1
FoxM1 repression during human aging leads to mitotic decline and aneuploidy-driven full senescence
Nature Communications·2018·134 cit.·DOI
2
Vascular Endothelial Growth Factor Receptor 2 (VEGFR-2) Plays a Key Role in Vasculogenic Mimicry Formation, Neovascularization and Tumor Initiation by Glioma Stem-like Cells
PLoS ONE·2013·130 cit.·DOI
3
Correction: Vascular Endothelial Growth Factor Receptor 2 (VEGFR-2) Plays a Key Role in Vasculogenic Mimicry Formation, Neovascularization and Tumor Initiation by Glioma Stem-like Cells
PLoS ONE·2013·25 cit.·DOI
4
Japanese encephalitis virus NS5 protein interacts with nucleolin to enhance the virus replication
Journal of Virology·2024·10 cit.·DOI
5
Impaired Fat Absorption from Intestinal Tract in High-Fat Diet Fed Male Mice Deficient in Proglucagon-Derived Peptides
Nutrients·2024·6 cit.·DOI
6
Lipoprotein(a)/CD36 Interaction Drives IL-6/RhoA-GTP Signaling and miRNA Epigenetic Regulation in Coronary Artery Spasm
Pharmaceuticals·2025·1 cit.·DOI
7
Caltubin regulates microtubule stability via Ca <sup>2+</sup> -dependent mechanisms favouring neurite regrowth
bioRxiv (Cold Spring Harbor Laboratory)·2023·DOI
8
Lipoprotein(a) Triggers CD36-Dependent IL-6/RhoA-GTP Axis Activation and Epigenetic Regulation of miRNA Expression in Coronary Artery Spasm
Preprints.org·2025·DOI
9
Spatial Transcriptome Analysis Reveals Factors Involved in Actinic Cheilosis Transformation to Squamous Cell Carcinoma of the Lip
Journal of Investigative Dermatology·2025·DOI
10
Molecular basis of mitotic decline during human aging
bioRxiv (Cold Spring Harbor Laboratory)·2018·DOI

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