Cell Signaling Technology

Cdc45 Antibody

#3673 · $317

100
Citing papers
12.7k
Combined citations
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Research areas

DNA Repair Mechanisms9Neuroinflammation and Neurodegeneration Mechanisms7Cell death mechanisms and regulation7Microtubule and mitosis dynamics6Cancer-related Molecular Pathways6Epigenetics and DNA Methylation6Mesenchymal stem cell research5Neurogenesis and neuroplasticity mechanisms5

Papers citing this product

1
Mutational signatures in esophageal adenocarcinoma define etiologically distinct subgroups with therapeutic relevance
Nature Genetics·2016·433 cit.·DOI
2
MicroRNA-223 Is Commonly Repressed in Hepatocellular Carcinoma and Potentiates Expression of Stathmin1
Gastroenterology·2008·422 cit.·DOI
3
Psychedelics promote plasticity by directly binding to BDNF receptor TrkB
Nature Neuroscience·2023·421 cit.·DOI
4
Chk1 Mediates S and G2 Arrests through Cdc25A Degradation in Response to DNA-damaging Agents
Journal of Biological Chemistry·2003·382 cit.·DOI
5
The Wnt5A/Protein Kinase C Pathway Mediates Motility in Melanoma Cells via the Inhibition of Metastasis Suppressors and Initiation of an Epithelial to Mesenchymal Transition
Journal of Biological Chemistry·2007·368 cit.·DOI
6
GABA-cAMP Response Element-Binding Protein Signaling Regulates Maturation and Survival of Newly Generated Neurons in the Adult Hippocampus
Journal of Neuroscience·2009·339 cit.·DOI
7
BRCA locus-specific loss of heterozygosity in germline BRCA1 and BRCA2 carriers
Nature Communications·2017·308 cit.·DOI
8
Activation of PI3K Is Indispensable for Interleukin 7–mediated Viability, Proliferation, Glucose Use, and Growth of T Cell Acute Lymphoblastic Leukemia Cells
The Journal of Experimental Medicine·2004·274 cit.·DOI
9
Minocycline Alleviates Death of Oligodendrocytes by Inhibiting Pro-Nerve Growth Factor Production in Microglia after Spinal Cord Injury
Journal of Neuroscience·2007·270 cit.·DOI
10
Estrogen Receptor-β Is Critical to Granulosa Cell Differentiation and the Ovulatory Response to Gonadotropins
Endocrinology·2005·259 cit.·DOI

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