Cell Signaling Technology

GAD1 Antibody

#5305 · $317

100
Citing papers
14.5k
Combined citations
Supplier page Find this product

Research areas

MicroRNA in disease regulation11Cell death mechanisms and regulation10NF-κB Signaling Pathways9Cancer-related Molecular Pathways8PI3K/AKT/mTOR signaling in cancer7Circadian rhythm and melatonin5Cytokine Signaling Pathways and Interactions5HER2/EGFR in Cancer Research5

Papers citing this product

1
Expression of p95HER2, a Truncated Form of the HER2 Receptor, and Response to Anti-HER2 Therapies in Breast Cancer
JNCI Journal of the National Cancer Institute·2007·876 cit.·DOI
2
Specific MicroRNAs Modulate Embryonic Stem Cell–Derived Neurogenesis
Stem Cells·2005·676 cit.·DOI
3
c-IAP1 and c-IAP2 Are Critical Mediators of Tumor Necrosis Factor α (TNFα)-induced NF-κB Activation
Journal of Biological Chemistry·2008·536 cit.·DOI
4
Elevated Levels of Cholesterol-Rich Lipid Rafts in Cancer Cells Are Correlated with Apoptosis Sensitivity Induced by Cholesterol-Depleting Agents
American Journal Of Pathology·2006·532 cit.·DOI
5
Rapid Akt activation by nicotine and a tobacco carcinogen modulates the phenotype of normal human airway epithelial cells
Journal of Clinical Investigation·2003·511 cit.·DOI
6
SARS-CoV-2 infects cells after viral entry via clathrin-mediated endocytosis
Journal of Biological Chemistry·2021·503 cit.·DOI
7
Rapid Akt activation by nicotine and a tobacco carcinogen modulates the phenotype of normal human airway epithelial cells
Journal of Clinical Investigation·2003·500 cit.·DOI
8
Targeting AKT/mTOR and ERK MAPK signaling inhibits hormone-refractory prostate cancer in a preclinical mouse model
Journal of Clinical Investigation·2008·387 cit.·DOI
9
Lapatinib (Tykerb, GW572016) Reverses Multidrug Resistance in Cancer Cells by Inhibiting the Activity of ATP-Binding Cassette Subfamily B Member 1 and G Member 2
Cancer Research·2008·384 cit.·DOI
10
HIF-1α-PDK1 axis-induced active glycolysis plays an essential role in macrophage migratory capacity
Nature Communications·2016·380 cit.·DOI

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