Cell Signaling Technology

Phospho-Ret (Tyr905) Antibody

#3221 · $362

100
Citing papers
15.3k
Combined citations
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Research areas

Autophagy in Disease and Therapy10Cell Adhesion Molecules Research6Genomics, phytochemicals, and oxidative stress5MicroRNA in disease regulation5Adipose Tissue and Metabolism4HER2/EGFR in Cancer Research4Cancer Cells and Metastasis4Cancer Genomics and Diagnostics4

Papers citing this product

1
Disrupted cardiac development but normal hematopoiesis in mice deficient in the second CXCL12/SDF-1 receptor, CXCR7
Proceedings of the National Academy of Sciences·2007·622 cit.·DOI
2
Klotho Inhibits Transforming Growth Factor-β1 (TGF-β1) Signaling and Suppresses Renal Fibrosis and Cancer Metastasis in Mice
Journal of Biological Chemistry·2011·530 cit.·DOI
3
Periostin is essential for cardiac healingafter acute myocardial infarction
The Journal of Experimental Medicine·2008·460 cit.·DOI
4
Microglial Stimulation of Glioblastoma Invasion Involves Epidermal Growth Factor Receptor (EGFR) and Colony Stimulating Factor 1 Receptor (CSF-1R) Signaling
Molecular Medicine·2012·443 cit.·DOI
5
β-Arrestin–mediated β1-adrenergic receptor transactivation of the EGFR confers cardioprotection
Journal of Clinical Investigation·2007·441 cit.·DOI
6
Autocrine VEGF–VEGFR2–Neuropilin-1 signaling promotes glioma stem-like cell viability and tumor growth
The Journal of Experimental Medicine·2012·402 cit.·DOI
7
Study of the Biologic Effects of Lapatinib, a Reversible Inhibitor of ErbB1 and ErbB2 Tyrosine Kinases, on Tumor Growth and Survival Pathways in Patients With Advanced Malignancies
Journal of Clinical Oncology·2005·364 cit.·DOI
8
Dectin-1 Stimulation by <i>Candida albicans</i> Yeast or Zymosan Triggers NFAT Activation in Macrophages and Dendritic Cells
The Journal of Immunology·2007·359 cit.·DOI
9
CD133/prominin-1 is a potential therapeutic target for antibody-drug conjugates in hepatocellular and gastric cancers
British Journal of Cancer·2008·289 cit.·DOI
10
WNT signaling drives cholangiocarcinoma growth and can be pharmacologically inhibited
Journal of Clinical Investigation·2015·274 cit.·DOI

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