Cell Signaling Technology

Bcl-2 (124) Mouse Monoclonal Antibody (SignalFlex™ Pacific Blue™ Conjugate)

#39223 · $458

26
Citing papers
1.2k
Combined citations
Supplier page Find this product

Research areas

Chronic Myeloid Leukemia Treatments8PI3K/AKT/mTOR signaling in cancer5Cancer, Hypoxia, and Metabolism5Eosinophilic Disorders and Syndromes4Cancer-related Molecular Pathways4Ubiquitin and proteasome pathways4Chronic Lymphocytic Leukemia Research3Neuroblastoma Research and Treatments3

Papers citing this product

1
Inhibition of NFκB Increases the Efficacy of Cisplatin in in Vitro and in Vivo Ovarian Cancer Models
Journal of Biological Chemistry·2004·212 cit.·DOI
2
Inhibition of Inhibitor of Nuclear Factor-κB Phosphorylation Increases the Efficacy of Paclitaxel in <b> <i>in Vitro</i> </b> and <b> <i>in Vivo</i> </b> Ovarian Cancer Models
Clinical Cancer Research·2004·114 cit.·DOI
3
Complex Regulation of the Cyclin-Dependent Kinase Inhibitor p27kip1 in Thyroid Cancer Cells by the PI3K/AKT Pathway
American Journal Of Pathology·2005·98 cit.·DOI
4
USP10 modulates the SKP2/Bcr-Abl axis via stabilizing SKP2 in chronic myeloid leukemia
Cell Discovery·2019·97 cit.·DOI
5
Kynurenic acid inhibits colon cancer proliferation in vitro: effects on signaling pathways
Amino Acids·2014·91 cit.·DOI
6
The mTOR inhibitor rapamycin down-regulates the expression of the ubiquitin ligase subunit Skp2 in breast cancer cells
Breast Cancer Research·2006·88 cit.·DOI
7
Anti-rheumatic agent auranofin induced apoptosis in chronic myeloid leukemia cells resistant to imatinib through both Bcr/Abl-dependent and -independent mechanisms
Oncotarget·2014·80 cit.·DOI
8
Aurora A Kinase Inhibitor AKI603 Induces Cellular Senescence in Chronic Myeloid Leukemia Cells Harboring T315I Mutation
Scientific Reports·2016·45 cit.·DOI
9
HIF2α reduces growth rate but promotes angiogenesis in a mouse model of neuroblastoma
BMC Cancer·2007·38 cit.·DOI
10
Activation of a prometastatic gene expression program in hypoxic neuroblastoma cells
Endocrine Related Cancer·2009·36 cit.·DOI

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