Cell Signaling Technology

UCHL3 Antibody

#3525 · western blot · $317

65
Evidence Score · Well-Established
Used by 184 independent labs across 200 papers. How we score this →
100
Citing papers
10.1k
Combined citations
Supplier page Find this product

Research areas

Mitochondrial Function and Pathology8MicroRNA in disease regulation7Immunotherapy and Immune Responses6Neurogenesis and neuroplasticity mechanisms6Neuropeptides and Animal Physiology5Adipose Tissue and Metabolism5Cancer, Hypoxia, and Metabolism5Ion Channels and Receptors4

Papers citing this product

1
Exosomes from M1-Polarized Macrophages Enhance Paclitaxel Antitumor Activity by Activating Macrophages-Mediated Inflammation
Theranostics·2019·482 cit.·DOI
2
ER sliding dynamics and ER–mitochondrial contacts occur on acetylated microtubules
The Journal of Cell Biology·2010·395 cit.·DOI
3
<p>A Nanodrug Consisting Of Doxorubicin And Exosome Derived From Mesenchymal Stem Cells For Osteosarcoma Treatment In Vitro</p>
International Journal of Nanomedicine·2019·311 cit.·DOI
4
Cancer Stem Cells Contribute to Cisplatin Resistance in <i>Brca1/p53</i> –Mediated Mouse Mammary Tumors
Cancer Research·2008·302 cit.·DOI
5
A <i>Toxoplasma</i> dense granule protein, GRA24, modulates the early immune response to infection by promoting a direct and sustained host p38 MAPK activation
The Journal of Experimental Medicine·2013·283 cit.·DOI
6
Phosphorylation of Serine 468 by GSK-3β Negatively Regulates Basal p65 NF-κB Activity
Journal of Biological Chemistry·2004·247 cit.·DOI
7
PKM2 coordinates glycolysis with mitochondrial fusion and oxidative phosphorylation
Protein & Cell·2019·246 cit.·DOI
8
Estrogen Stimulates Postsynaptic Density-95 Rapid Protein Synthesis via the Akt/Protein Kinase B Pathway
Journal of Neuroscience·2003·196 cit.·DOI
9
Loss of HRD1-Mediated Protein Degradation Causes Amyloid Precursor Protein Accumulation and Amyloid-β Generation
Journal of Neuroscience·2010·186 cit.·DOI
10
Fibronectin-guided migration of carcinoma collectives
Nature Communications·2017·182 cit.·DOI

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