Cell Signaling Technology

PhosphoPlus® p38 MAPK (Thr180/Tyr182) Antibody Duet

#8203 · $730

100
Citing papers
6.3k
Combined citations
Supplier page Find this product

Research areas

Immune Cell Function and Interaction8Cancer, Hypoxia, and Metabolism8Cancer-related molecular mechanisms research6Growth Hormone and Insulin-like Growth Factors6Immune cells in cancer5Protein Kinase Regulation and GTPase Signaling5Circular RNAs in diseases5Immune Response and Inflammation4

Papers citing this product

1
Whole-exome sequencing of pancreatic cancer defines genetic diversity and therapeutic targets
Nature Communications·2015·1.1k cit.·DOI
2
Differential requirement for the activation of the inflammasome for processing and release of IL-1β in monocytes and macrophages
Blood·2008·777 cit.·DOI
3
Oncogenic Transformation by Inhibitor-Sensitive and -Resistant EGFR Mutants
PLoS Medicine·2005·683 cit.·DOI
4
Dectin-1 Stimulation by <i>Candida albicans</i> Yeast or Zymosan Triggers NFAT Activation in Macrophages and Dendritic Cells
The Journal of Immunology·2007·359 cit.·DOI
5
Modeling Colon Adenocarcinomas in Vitro
American Journal Of Pathology·2011·176 cit.·DOI
6
Regulation of Hypoxia-Inducible Factor-1α, Vascular Endothelial Growth Factor, and Angiogenesis by an Insulin-Like Growth Factor-I Receptor Autocrine Loop in Human Pancreatic Cancer
American Journal Of Pathology·2003·175 cit.·DOI
7
Activation of Metabotropic Glutamate Receptor 5 in the Amygdala Modulates Pain-Like Behavior
Journal of Neuroscience·2010·130 cit.·DOI
8
Superior Activity of the Combination of Histone Deacetylase Inhibitor LAQ824 and the FLT-3 Kinase Inhibitor PKC412 against Human Acute Myelogenous Leukemia Cells with Mutant FLT-3
Clinical Cancer Research·2004·125 cit.·DOI
9
P2X<sub>7</sub> receptors stimulate AKT phosphorylation in astrocytes
British Journal of Pharmacology·2004·121 cit.·DOI
10
Genome-wide CRISPR screen identifies FAM49B as a key regulator of actin dynamics and T cell activation
Proceedings of the National Academy of Sciences·2018·120 cit.·DOI