Cell Signaling Technology

Bcl-3 (E4D4K) Rabbit Monoclonal Antibody

#16611 · $161

26
Citing papers
752
Combined citations
Supplier page Find this product

Research areas

Immune Cell Function and Interaction3Wnt/β-catenin signaling in development and cancer2Cellular transport and secretion2Immune Response and Inflammation2Immunotherapy and Immune Responses2Cancer Cells and Metastasis1Atherosclerosis and Cardiovascular Diseases1Advanced biosensing and bioanalysis techniques1

Papers citing this product

1
CaMK4-dependent activation of AKT/mTOR and CREM-α underlies autoimmunity-associated Th17 imbalance
Journal of Clinical Investigation·2014·216 cit.·DOI
2
Nur77 suppresses hepatocellular carcinoma via switching glucose metabolism toward gluconeogenesis through attenuating phosphoenolpyruvate carboxykinase sumoylation
Nature Communications·2017·135 cit.·DOI
3
Stemness, Pluripotentiality, and Wnt Antagonism: sFRP4, a Wnt antagonist Mediates Pluripotency and Stemness in Glioblastoma
Cancers·2018·70 cit.·DOI
4
MBD2 upregulates miR-301a-5p to induce kidney cell apoptosis during vancomycin-induced AKI
Cell Death and Disease·2017·68 cit.·DOI
5
Expression of IGF/insulin receptor in prostate cancer tissue and progression to lethal disease
Carcinogenesis·2018·59 cit.·DOI
6
Human Langerhans cells use an IL-15R-α/IL-15/pSTAT5-dependent mechanism to break T-cell tolerance against the self-differentiation tumor antigen WT1
Blood·2012·51 cit.·DOI
7
Vasculoprotective Effects of 3-Hydroxybenzaldehyde against VSMCs Proliferation and ECs Inflammation
PLoS ONE·2016·39 cit.·DOI
8
Inhibition of cAMP-Dependent PKA Activates β2-Adrenergic Receptor Stimulation of Cytosolic Phospholipase A2 via Raf-1/MEK/ERK and IP3-Dependent Ca2+ Signaling in Atrial Myocytes
PLoS ONE·2016·19 cit.·DOI
9
Anti-ovarian tumor response of donor peripheral blood mononuclear cells is due to infiltrating cytotoxic NK cells
Oncotarget·2016·18 cit.·DOI
10
Ftr82 Is Critical for Vascular Patterning during Zebrafish Development
International Journal of Molecular Sciences·2017·14 cit.·DOI

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