Cell Signaling Technology

Caspase-9 (C9) Mouse Monoclonal Antibody

#9508 · $148,348

65

Evidence Score · Well-Established

Used by 374 independent labs across 399 papers. How we score this →

100

Citing papers

9.4k

Combined citations

Supplier page Find this product

Research areas

PI3K/AKT/mTOR signaling in cancer11 Cell death mechanisms and regulation11 MicroRNA in disease regulation9 Autophagy in Disease and Therapy9 Epigenetics and DNA Methylation8 Circular RNAs in diseases7 Cancer-related Molecular Pathways7 Cancer, Hypoxia, and Metabolism7

Papers citing this product

1

AIM2 and NLRP3 inflammasomes activate both apoptotic and pyroptotic death pathways via ASC

Cell Death and Differentiation·2013·495 cit.·DOI

2

DNA-methylation-mediated activating of lncRNA SNHG12 promotes temozolomide resistance in glioblastoma

Molecular Cancer·2020·346 cit.·DOI

3

Targeting lonidamine to mitochondria mitigates lung tumorigenesis and brain metastasis

Nature Communications·2019·242 cit.·DOI

4

Myeloid-derived growth factor (C19orf10) mediates cardiac repair following myocardial infarction

Nature Medicine·2015·216 cit.·DOI

5

mTOR signaling regulates central and peripheral circadian clock function

PLoS Genetics·2018·199 cit.·DOI

6

A short G1 phase imposes constitutive replication stress and fork remodelling in mouse embryonic stem cells

Nature Communications·2016·198 cit.·DOI

7

Epicardial YAP/TAZ orchestrate an immunosuppressive response following myocardial infarction

Journal of Clinical Investigation·2017·176 cit.·DOI

8

LPCAT1 promotes brain metastasis of lung adenocarcinoma by up-regulating PI3K/AKT/MYC pathway

Journal of Experimental & Clinical Cancer Research·2019·164 cit.·DOI

9

Cross-Talk between NFkB and the PI3-Kinase/AKT Pathway Can Be Targeted in Primary Effusion Lymphoma (PEL) Cell Lines for Efficient Apoptosis

PLoS ONE·2012·152 cit.·DOI

10

Alpha-enolase regulates the malignant phenotype of pulmonary artery smooth muscle cells via the AMPK-Akt pathway

Nature Communications·2018·148 cit.·DOI

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