Cell Signaling Technology

14-3-3 epsilon Antibody

#9635 · $317

100
Citing papers
4.9k
Combined citations
Supplier page Find this product

Research areas

Adipose Tissue and Metabolism22Cancer, Hypoxia, and Metabolism17Adipokines, Inflammation, and Metabolic Diseases8PI3K/AKT/mTOR signaling in cancer7Metabolism, Diabetes, and Cancer7Protein Tyrosine Phosphatases7Mitochondrial Function and Pathology614-3-3 protein interactions6

Papers citing this product

1
The transcription factor HIF-1α plays a critical role in the growth factor-dependent regulation of both aerobic and anaerobic glycolysis
Genes & Development·2007·422 cit.·DOI
2
D1 Dopamine Receptor Supersensitivity in the Dopamine-Depleted Striatum Results from a Switch in the Regulation of ERK1/2/MAP Kinase
Journal of Neuroscience·2002·367 cit.·DOI
3
Flexible patch with printable and antibacterial conductive hydrogel electrodes for accelerated wound healing
Biomaterials·2022·198 cit.·DOI
4
Human adipose beiging in response to cold and mirabegron
JCI Insight·2018·193 cit.·DOI
5
METRNL attenuates lipid-induced inflammation and insulin resistance via AMPK or PPARδ-dependent pathways in skeletal muscle of mice
Experimental & Molecular Medicine·2018·178 cit.·DOI
6
Perinatal Exposure of Mice to the Pesticide DDT Impairs Energy Expenditure and Metabolism in Adult Female Offspring
PLoS ONE·2014·175 cit.·DOI
7
Phosphatidylinositol 3-Kinase/Akt Signaling Is Neither Required for Hypoxic Stabilization of HIF-1α nor Sufficient for HIF-1-dependent Target Gene Transcription
Journal of Biological Chemistry·2002·171 cit.·DOI
8
Bacteria-Induced Uroplakin Signaling Mediates Bladder Response to Infection
PLoS Pathogens·2009·170 cit.·DOI
9
Ctr1 Is an Apical Copper Transporter in Mammalian Intestinal Epithelial Cells in Vivo That Is Controlled at the Level of Protein Stability
Journal of Biological Chemistry·2010·154 cit.·DOI
10
Fibroblast growth factor-21 enhances mitochondrial functions and increases the activity of PGC-1α in human dopaminergic neurons via Sirtuin-1
SpringerPlus·2014·116 cit.·DOI

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