Cell Signaling Technology

Phospho-MARCKS (Ser159/163) (D13D2) Rabbit Monoclonal Antibody

#11992 · $176

100
Citing papers
5.8k
Combined citations
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Research areas

Neuroinflammation and Neurodegeneration Mechanisms14Amyotrophic Lateral Sclerosis Research12Neurogenesis and neuroplasticity mechanisms8Nerve injury and regeneration6Neurogenetic and Muscular Disorders Research6Alzheimer's disease research and treatments6Immune Cell Function and Interaction6Signaling Pathways in Disease6

Papers citing this product

1
Neuron-released oligomeric α-synuclein is an endogenous agonist of TLR2 for paracrine activation of microglia
Nature Communications·2013·838 cit.·DOI
2
Inhibition of Microglial Phagocytosis Is Sufficient To Prevent Inflammatory Neuronal Death
The Journal of Immunology·2011·372 cit.·DOI
3
Nucleocytoplasmic Shuttling of HMGB1 Is Regulated by Phosphorylation That Redirects It toward Secretion
The Journal of Immunology·2006·362 cit.·DOI
4
Microglial p38α MAPK is a key regulator of proinflammatory cytokine up-regulation induced by toll-like receptor (TLR) ligands or beta-amyloid (Aβ)
Journal of Neuroinflammation·2011·283 cit.·DOI
5
MFG‐E8 reverses microglial‐induced neurotoxic astrocyte (A1) via NF‐κB and PI3K‐Akt pathways
Journal of Cellular Physiology·2018·190 cit.·DOI
6
GM-CSF increases LPS-induced production of proinflammatory mediators via upregulation of TLR4 and CD14 in murine microglia
Journal of Neuroinflammation·2012·147 cit.·DOI
7
The c-Jun N-Terminal Kinase Activator Dual Leucine Zipper Kinase Regulates Axon Growth and Neuronal Migration in the Developing Cerebral Cortex
Journal of Neuroscience·2006·145 cit.·DOI
8
Intracellular Proteolysis of Progranulin Generates Stable, Lysosomal Granulins that Are Haploinsufficient in Patients with Frontotemporal Dementia Caused by<i>GRN</i>Mutations
eNeuro·2017·137 cit.·DOI
9
The p38α MAPK Regulates Microglial Responsiveness to Diffuse Traumatic Brain Injury
Journal of Neuroscience·2013·127 cit.·DOI
10
Characterization of Sparstolonin B, a Chinese Herb-derived Compound, as a Selective Toll-like Receptor Antagonist with Potent Anti-inflammatory Properties
Journal of Biological Chemistry·2011·126 cit.·DOI