Cell Signaling Technology

TIAR (D26E4) Rabbit Monoclonal Antibody

#8611 · $161,351

65
Evidence Score · Well-Established
Used by 307 independent labs across 321 papers. How we score this →
100
Citing papers
4.1k
Combined citations
Supplier page Find this product

Research areas

Receptor Mechanisms and Signaling8Cellular transport and secretion7PI3K/AKT/mTOR signaling in cancer5Cancer-related gene regulation5Heat shock proteins research5Protein Kinase Regulation and GTPase Signaling5Cancer, Hypoxia, and Metabolism5Cancer Research and Treatments4

Papers citing this product

1
DAP-kinase is a mediator of endoplasmic reticulum stress-induced caspase activation and autophagic cell death
Cell Death and Differentiation·2008·251 cit.·DOI
2
Wnt Isoform-Specific Interactions with Coreceptor Specify Inhibition or Potentiation of Signaling by LRP6 Antibodies
PLoS ONE·2010·204 cit.·DOI
3
miR-135b mediates NPM-ALK–driven oncogenicity and renders IL-17–producing immunophenotype to anaplastic large cell lymphoma
Blood·2011·181 cit.·DOI
4
Metformin Down-regulates TNF-α Secretion via Suppression of Scavenger Receptors in Macrophages
Immune Network·2013·155 cit.·DOI
5
Phosphorylation of EEA1 by p38 MAP kinase regulates μ opioid receptor endocytosis
The EMBO Journal·2005·138 cit.·DOI
6
Oxidative stress-mediated mitochondrial dysfunction facilitates mesenchymal stem cell senescence in ankylosing spondylitis
Cell Death and Disease·2020·112 cit.·DOI
7
FGL1 regulates acquired resistance to Gefitinib by inhibiting apoptosis in non-small cell lung cancer
Respiratory Research·2020·112 cit.·DOI
8
Brain-Derived Neurotrophic Factor Ameliorates Learning Deficits in a Rat Model of Alzheimer's Disease Induced by Aβ1-42
PLoS ONE·2015·106 cit.·DOI
9
Heat Shock Protein 90 Mediates Efficient Antigen Cross Presentation through the Scavenger Receptor Expressed by Endothelial Cells-I
The Journal of Immunology·2010·106 cit.·DOI
10
Cell Growth Inhibition and Functioning of Human Somatostatin Receptor Type 2 Are Modulated by Receptor Heterodimerization
Molecular Endocrinology·2008·103 cit.·DOI

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