Cell Signaling Technology

PTMScan® Ubiquitin Remnant Motif (K-epsilon-GG) Kit

#5562 · mass spectrometry · $2,540

60
Evidence Score · Well-Established
Used by 93 independent labs across 100 papers. How we score this →
100
Citing papers
8.9k
Combined citations
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Research areas

Alzheimer's disease research and treatments14Ubiquitin and proteasome pathways9Neuroscience and Neuropharmacology Research8Cancer Immunotherapy and Biomarkers7Autophagy in Disease and Therapy7Cancer Cells and Metastasis6Microtubule and mitosis dynamics5Mitochondrial Function and Pathology5

Papers citing this product

1
Cancer-associated fibroblasts induce antigen-specific deletion of CD8 + T Cells to protect tumour cells
Nature Communications·2018·558 cit.·DOI
2
Up-Regulation of Phosphorylated/Activated p70 S6 Kinase and Its Relationship to Neurofibrillary Pathology in Alzheimer's Disease
American Journal Of Pathology·2003·314 cit.·DOI
3
Effects of Body Temperature on Neural Activity in the Hippocampus: Regulation of Resting Membrane Potentials by Transient Receptor Potential Vanilloid 4
Journal of Neuroscience·2007·301 cit.·DOI
4
Hepatic carcinoma-associated fibroblasts induce IDO-producing regulatory dendritic cells through IL-6-mediated STAT3 activation
Oncogenesis·2016·259 cit.·DOI
5
Reconsolidation and Extinction Are Dissociable and Mutually Exclusive Processes: Behavioral and Molecular Evidence
Journal of Neuroscience·2014·251 cit.·DOI
6
Activation of B‐Raf kinase requires phosphorylation of the conserved residues Thr598 and Ser601
The EMBO Journal·2000·245 cit.·DOI
7
Insulin Dysfunction Induces<i>In Vivo</i>Tau Hyperphosphorylation through Distinct Mechanisms
Journal of Neuroscience·2007·236 cit.·DOI
8
The E3 ubiquitin ligase and RNA-binding protein ZNF598 orchestrates ribosome quality control of premature polyadenylated mRNAs
Nature Communications·2017·233 cit.·DOI
9
MAPT/Tau accumulation represses autophagy flux by disrupting IST1-regulated ESCRT-III complex formation: a vicious cycle in Alzheimer neurodegeneration
Autophagy·2019·210 cit.·DOI
10
Tumor Necrosis Factor (TNF) Receptor Shedding Controls Thresholds of Innate Immune Activation That Balance Opposing TNF Functions in Infectious and Inflammatory Diseases
The Journal of Experimental Medicine·2004·197 cit.·DOI

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