MedChemExpress

Liproxstatin-1

#HY-12726

30
Citing papers
1.3k
Combined citations
Supplier page Find this product

Research areas

Ferroptosis and cancer prognosis17Cancer, Lipids, and Metabolism6RNA modifications and cancer6Cancer-related molecular mechanisms research5Immune cells in cancer4Extracellular vesicles in disease3Autophagy in Disease and Therapy3Inflammasome and immune disorders3

Papers citing this product

1
A photo-triggered self-accelerated nanoplatform for multifunctional image-guided combination cancer immunotherapy
Nature Communications·2023·178 cit.·DOI
2
The interaction between STING and NCOA4 exacerbates lethal sepsis by orchestrating ferroptosis and inflammatory responses in macrophages
Cell Death and Disease·2022·171 cit.·DOI
3
Targeting de novo lipogenesis and the Lands cycle induces ferroptosis in KRAS-mutant lung cancer
Nature Communications·2022·168 cit.·DOI
4
Inhibition of CDK1 Overcomes Oxaliplatin Resistance by Regulating ACSL4‐mediated Ferroptosis in Colorectal Cancer
Advanced Science·2023·148 cit.·DOI
5
Ferroportin‐Dependent Iron Homeostasis Protects against Oxidative Stress‐Induced Nucleus Pulposus Cell Ferroptosis and Ameliorates Intervertebral Disc Degeneration <i>In Vivo</i>
Oxidative Medicine and Cellular Longevity·2021·127 cit.·DOI
6
Nanoparticle delivery of miR-21-3p sensitizes melanoma to anti-PD-1 immunotherapy by promoting ferroptosis
Journal for ImmunoTherapy of Cancer·2022·121 cit.·DOI
7
Olaparib synergizes with arsenic trioxide by promoting apoptosis and ferroptosis in platinum-resistant ovarian cancer
Cell Death and Disease·2022·63 cit.·DOI
8
PRMT3‐Mediated Arginine Methylation of METTL14 Promotes Malignant Progression and Treatment Resistance in Endometrial Carcinoma
Advanced Science·2023·60 cit.·DOI
9
Ferroptotic MSCs protect mice against sepsis via promoting macrophage efferocytosis
Cell Death and Disease·2022·36 cit.·DOI
10
Curcumin promotes ferroptosis in hepatocellular carcinoma via upregulation of ACSL4
Journal of Cancer Research and Clinical Oncology·2024·30 cit.·DOI

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