Cell Signaling Technology

Tri-Methyl-Histone H3 (Lys36) Antibody

#9763 · $362

100
Citing papers
4.3k
Combined citations
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Research areas

Cancer-related molecular mechanisms research10TGF-β signaling in diseases9Nerve injury and regeneration9Spinal Cord Injury Research9MicroRNA in disease regulation8Circular RNAs in diseases5Bone Metabolism and Diseases5Virus-based gene therapy research5

Papers citing this product

1
Umbilical Cord-Derived Mesenchymal Stem Cell-Derived Exosomal MicroRNAs Suppress Myofibroblast Differentiation by Inhibiting the Transforming Growth Factor-β/SMAD2 Pathway During Wound Healing
Stem Cells Translational Medicine·2016·596 cit.·DOI
2
Synthetic lethality between androgen receptor signalling and the PARP pathway in prostate cancer
Nature Communications·2017·278 cit.·DOI
3
miR-30 Family Members Negatively Regulate Osteoblast Differentiation
Journal of Biological Chemistry·2012·185 cit.·DOI
4
Pten Deletion Promotes Regrowth of Corticospinal Tract Axons 1 Year after Spinal Cord Injury
Journal of Neuroscience·2015·171 cit.·DOI
5
Transneuronal delivery of hyper-interleukin-6 enables functional recovery after severe spinal cord injury in mice
Nature Communications·2021·128 cit.·DOI
6
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components
Cell Communication and Signaling·2020·127 cit.·DOI
7
Endoglin Requirement for BMP9 Signaling in Endothelial Cells Reveals New Mechanism of Action for Selective Anti-Endoglin Antibodies
PLoS ONE·2012·110 cit.·DOI
8
Dynamic smad‐mediated BMP signaling revealed through transgenic zebrafish
Developmental Dynamics·2011·102 cit.·DOI
9
Functional analysis of the BMP9 response of ALK1 mutants from HHT2 patients: a diagnostic tool for novel ACVRL1 mutations
Blood·2010·95 cit.·DOI
10
Blockade of Autophagy Prevents the Progression of Hyperuricemic Nephropathy Through Inhibiting NLRP3 Inflammasome-Mediated Pyroptosis
Frontiers in Immunology·2022·87 cit.·DOI

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