Cell Signaling Technology

PathScan® Phospho-p44/42 MAPK (Thr202/Tyr204) Sandwich ELISA Antibody Pair

#7246 · $553

100
Citing papers
7.2k
Combined citations
Supplier page Find this product

Research areas

Cancer-related molecular mechanisms research10Autophagy in Disease and Therapy9Cell death mechanisms and regulation6PI3K/AKT/mTOR signaling in cancer6MicroRNA in disease regulation6Tea Polyphenols and Effects6Immune cells in cancer5Neurogenesis and neuroplasticity mechanisms5

Papers citing this product

1
A Constituent of Green Tea, Epigallocatechin-3-gallate, Activates Endothelial Nitric Oxide Synthase by a Phosphatidylinositol-3-OH-kinase-, cAMP-dependent Protein Kinase-, and Akt-dependent Pathway and Leads to Endothelial-dependent Vasorelaxation
Journal of Biological Chemistry·2004·332 cit.·DOI
2
Release of SOS2 kinase from sequestration with GIGANTEA determines salt tolerance in Arabidopsis
Nature Communications·2013·321 cit.·DOI
3
hsa_circ_0013958: a circular <scp>RNA</scp> and potential novel biomarker for lung adenocarcinoma
FEBS Journal·2017·303 cit.·DOI
4
Mitochondrial RNA modifications shape metabolic plasticity in metastasis
Nature·2022·289 cit.·DOI
5
Spatially resolved transcriptomics reveals the architecture of the tumor-microenvironment interface
Nature Communications·2021·275 cit.·DOI
6
Pro-oxidative activities and dose–response relationship of (−)-epigallocatechin-3-gallate in the inhibition of lung cancer cell growth: a comparative study in vivo and in vitro
Carcinogenesis·2010·237 cit.·DOI
7
The c-Jun N-Terminal Protein Kinase Signaling Pathway Mediates Bax Activation and Subsequent Neuronal Apoptosis through Interaction with Bim after Transient Focal Cerebral Ischemia
Journal of Neuroscience·2004·233 cit.·DOI
8
Induction of lysosomal and mitochondrial biogenesis by AMPK phosphorylation of FNIP1
Science·2023·197 cit.·DOI
9
Knowledge-Based Configuration: From Research to Business Cases
International Journal of Molecular Sciences·2014·186 cit.·DOI
10
Transforming Growth Factor-β Differentially Inhibits MyD88-dependent, but Not TRAM- and TRIF-dependent, Lipopolysaccharide-induced TLR4 Signaling
Journal of Biological Chemistry·2004·185 cit.·DOI

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