Cell Signaling Technology

hnRNP U (E2O1R) Rabbit Monoclonal Antibody

#34095 · $161

100
Citing papers
7.8k
Combined citations
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Research areas

Ubiquitin and proteasome pathways6Inflammasome and immune disorders6Epigenetics and DNA Methylation6Hippo pathway signaling and YAP/TAZ5Autophagy in Disease and Therapy5Cancer-related molecular mechanisms research5Metabolism, Diabetes, and Cancer5Immune cells in cancer4

Papers citing this product

1
A systems study reveals concurrent activation of AMPK and mTOR by amino acids
Nature Communications·2016·446 cit.·DOI
2
The Caspase-1 Digestome Identifies the Glycolysis Pathway as a Target during Infection and Septic Shock
Journal of Biological Chemistry·2007·323 cit.·DOI
3
NUPR1 is a critical repressor of ferroptosis
Nature Communications·2021·297 cit.·DOI
4
Starvation induces rapid degradation of selective autophagy receptors by endosomal microautophagy
The Journal of Cell Biology·2018·277 cit.·DOI
5
PINK1 and PARK2 Suppress Pancreatic Tumorigenesis through Control of Mitochondrial Iron-Mediated Immunometabolism
Developmental Cell·2018·257 cit.·DOI
6
Synthetic RNA–protein modules integrated with native translation mechanisms to control gene expression in malaria parasites
Nature Communications·2016·226 cit.·DOI
7
Moderate-Intensity Exercise and High-Intensity Interval Training Affect Insulin Sensitivity Similarly in Obese Adults
The Journal of Clinical Endocrinology & Metabolism·2020·195 cit.·DOI
8
Curcumin derivative WZ35 inhibits tumor cell growth via ROS-YAP-JNK signaling pathway in breast cancer
Journal of Experimental & Clinical Cancer Research·2019·194 cit.·DOI
9
Glucagon-Like Peptide-1 (GLP-1) Analog Liraglutide Inhibits Endothelial Cell Inflammation through a Calcium and AMPK Dependent Mechanism
PLoS ONE·2014·176 cit.·DOI
10
Estrogenic G protein‐coupled receptor 30 signaling is involved in regulation of endometrial carcinoma by promoting proliferation, invasion potential, and interleukin‐6 secretion via the MEK/ERK mitogen‐activated protein kinase pathway
Cancer Science·2009·155 cit.·DOI