Cell Signaling Technology

COL1A1 (E8F4L) Rabbit Monoclonal Antibody

#72026 · $180,996

71
Evidence Score · Well-Established
Used by 259 independent labs across 306 papers. How we score this →
100
Citing papers
1.4k
Combined citations
Supplier page Find this product

Research areas

Liver Disease Diagnosis and Treatment15Liver physiology and pathology11Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis10Extracellular vesicles in disease6Neonatal Respiratory Health Research6Chronic Kidney Disease and Diabetes5Cancer Cells and Metastasis5Fibroblast Growth Factor Research5

Papers citing this product

1
Parabacteroides distasonis ameliorates hepatic fibrosis potentially via modulating intestinal bile acid metabolism and hepatocyte pyroptosis in male mice
Nature Communications·2023·208 cit.·DOI
2
Dynamic and adaptive cancer stem cell population admixture in colorectal neoplasia
Cell stem cell·2022·113 cit.·DOI
3
Doxazosin Attenuates Liver Fibrosis by Inhibiting Autophagy in Hepatic Stellate Cells via Activation of the PI3K/Akt/mTOR Signaling Pathway
Drug Design Development and Therapy·2021·79 cit.·DOI
4
Notch-mediated hepatocyte MCP-1 secretion causes liver fibrosis
JCI Insight·2023·74 cit.·DOI
5
Dihydroquercetin Attenuates Silica-Induced Pulmonary Fibrosis by Inhibiting Ferroptosis Signaling Pathway
Frontiers in Pharmacology·2022·74 cit.·DOI
6
Motion lubrication suppressed mechanical activation via hydrated fibrous gene patch for tendon healing
Science Advances·2023·67 cit.·DOI
7
MALAT1 promotes liver fibrosis by sponging miR‑181a and activating TLR4‑NF‑κB signaling
International Journal of Molecular Medicine·2021·47 cit.·DOI
8
Locationally activated PRP via an injectable dual-network hydrogel for endometrial regeneration
Biomaterials·2024·43 cit.·DOI
9
Cell-intrinsic effects of clonal hematopoiesis in heart failure
Nature Cardiovascular Research·2023·38 cit.·DOI
10
Exosomes secreted by palmitic acid-treated hepatocytes promote LX-2 cell activation by transferring miRNA-107
Cell Death Discovery·2021·35 cit.·DOI

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