Cell Signaling Technology

Docetaxel

#9886 · $75

100
Citing papers
5.1k
Combined citations
Supplier page Find this product

Research areas

Genomics, phytochemicals, and oxidative stress10Cancer-related molecular mechanisms research7MicroRNA in disease regulation7Genetics and Neurodevelopmental Disorders7RNA modifications and cancer7Genomics and Chromatin Dynamics7Liver physiology and pathology6Connexins and lens biology6

Papers citing this product

1
Mesenchymal stem cell-derived exosomes promote hepatic regeneration in drug-induced liver injury models
Stem Cell Research & Therapy·2014·559 cit.·DOI
2
A genetic screen identifies the Triple T complex required for DNA damage signaling and ATM and ATR stability
Genes & Development·2010·214 cit.·DOI
3
The Neuroprotection of Liraglutide Against Ischaemia-induced Apoptosis through the Activation of the PI3K/AKT and MAPK Pathways
Scientific Reports·2016·179 cit.·DOI
4
Overexpression of CXCL5 Is Associated With Poor Survival in Patients With Pancreatic Cancer
American Journal Of Pathology·2011·178 cit.·DOI
5
KLF8 promotes human breast cancer cell invasion and metastasis by transcriptional activation of MMP9
Oncogene·2010·163 cit.·DOI
6
Protective Effects of Licochalcone A Ameliorates Obesity and Non-Alcoholic Fatty Liver Disease Via Promotion of the Sirt-1/AMPK Pathway in Mice Fed a High-Fat Diet
Cells·2019·152 cit.·DOI
7
Alu-dependent RNA editing of GLI1 promotes malignant regeneration in multiple myeloma
Nature Communications·2017·142 cit.·DOI
8
PTP1B inhibition suggests a therapeutic strategy for Rett syndrome
Journal of Clinical Investigation·2015·116 cit.·DOI
9
TCF12 Protein Functions as Transcriptional Repressor of E-cadherin, and Its Overexpression Is Correlated with Metastasis of Colorectal Cancer
Journal of Biological Chemistry·2011·113 cit.·DOI
10
Decreased mTOR signaling pathway in human idiopathic autism and in rats exposed to valproic acid
Acta Neuropathologica Communications·2015·108 cit.·DOI

Rate this reagent

Used this in your lab? Leave a quick rating. Reviews are anonymous and verified by academic email before they appear.

Your email is never shown publicly.