Cell Signaling Technology

Ribosomal Protein L13a Antibody

#2765 · $317

63
Evidence Score · Well-Established
Used by 99 independent labs across 100 papers. How we score this →
100
Citing papers
25k
Combined citations
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Research areas

Autophagy in Disease and Therapy13MicroRNA in disease regulation9interferon and immune responses8DNA Repair Mechanisms8Endoplasmic Reticulum Stress and Disease8Immune Cell Function and Interaction6Mitochondrial Function and Pathology6Inflammasome and immune disorders5

Papers citing this product

1
Nrf2 suppresses macrophage inflammatory response by blocking proinflammatory cytokine transcription
Nature Communications·2016·1.8k cit.·DOI
2
Mitochondrial membrane potential regulates PINK1 import and proteolytic destabilization by PARL
The Journal of Cell Biology·2010·1.3k cit.·DOI
3
Tumor-suppressive <i>miR-34a</i> induces senescence-like growth arrest through modulation of the E2F pathway in human colon cancer cells
Proceedings of the National Academy of Sciences·2007·902 cit.·DOI
4
Multi-omics analysis identifies ATF4 as a key regulator of the mitochondrial stress response in mammals
The Journal of Cell Biology·2017·835 cit.·DOI
5
FYCO1 is a Rab7 effector that binds to LC3 and PI3P to mediate microtubule plus end–directed vesicle transport
The Journal of Cell Biology·2010·639 cit.·DOI
6
Identification of the PANoptosome: A Molecular Platform Triggering Pyroptosis, Apoptosis, and Necroptosis (PANoptosis)
Frontiers in Cellular and Infection Microbiology·2020·530 cit.·DOI
7
A2A receptor signaling promotes peripheral tolerance by inducing T-cell anergy and the generation of adaptive regulatory T cells
Blood·2007·496 cit.·DOI
8
The dynamic interaction of AMBRA1 with the dynein motor complex regulates mammalian autophagy
The Journal of Cell Biology·2010·488 cit.·DOI
9
RETRACTED ARTICLE: A mechanism for the suppression of homologous recombination in G1 cells
Nature·2015·476 cit.·DOI
10
Insulin resistance causes inflammation in adipose tissue
Journal of Clinical Investigation·2018·464 cit.·DOI

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