Cell Signaling Technology

Phospho-SMAD3 (Ser423/425) (C25A9) Rabbit Monoclonal Antibody

#9520 · $169

100
Citing papers
12.2k
Combined citations
Supplier page Find this product

Research areas

TGF-β signaling in diseases16Cancer Cells and Metastasis12Cancer-related molecular mechanisms research11MicroRNA in disease regulation8Bone Metabolism and Diseases6Epigenetics and DNA Methylation6Circular RNAs in diseases6Kruppel-like factors research5

Papers citing this product

1
Post-transcriptional Regulation of Low Density Lipoprotein Receptor Protein by Proprotein Convertase Subtilisin/Kexin Type 9a in Mouse Liver
Journal of Biological Chemistry·2004·501 cit.·DOI
2
Lipopolysaccharide activation of the MEK-ERK1/2 pathway in human monocytic cells mediates tissue factor and tumor necrosis factor α expression by inducing Elk-1 phosphorylation and Egr-1 expression
Blood·2001·368 cit.·DOI
3
Sterol-dependent regulation of proprotein convertase subtilisin/kexin type 9 expression by sterol-regulatory element binding protein-2
Journal of Lipid Research·2007·349 cit.·DOI
4
The pro-metastasis effect of circANKS1B in breast cancer
Molecular Cancer·2018·347 cit.·DOI
5
Divergent effects of glucose and fructose on hepatic lipogenesis and insulin signaling
Journal of Clinical Investigation·2017·340 cit.·DOI
6
TGFβ2-induced formation of lipid droplets supports acidosis-driven EMT and the metastatic spreading of cancer cells
Nature Communications·2020·318 cit.·DOI
7
Polyunsaturated fatty acids suppress glycolytic and lipogenic genes through the inhibition of ChREBP nuclear protein translocation
Journal of Clinical Investigation·2005·314 cit.·DOI
8
Silencing long non-coding RNA Kcnq1ot1 alleviates pyroptosis and fibrosis in diabetic cardiomyopathy
Cell Death and Disease·2018·279 cit.·DOI
9
Overexpression of Insig-1 in the livers of transgenic mice inhibits SREBP processing and reduces insulin-stimulated lipogenesis
Journal of Clinical Investigation·2004·265 cit.·DOI
10
CCL2/CCR2 Chemokine Signaling Coordinates Survival and Motility of Breast Cancer Cells through Smad3 Protein- and p42/44 Mitogen-activated Protein Kinase (MAPK)-dependent Mechanisms
Journal of Biological Chemistry·2012·202 cit.·DOI

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