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Cell Signaling Technology
Caspase-3 Control Cell Extracts
#9663 · $165
100
Citing papers
3.2k
Combined citations
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Research areas
TGF-β signaling in diseases
8
Wnt/β-catenin signaling in development and cancer
6
Autophagy in Disease and Therapy
6
Cancer, Hypoxia, and Metabolism
5
Epigenetics and DNA Methylation
5
Cell death mechanisms and regulation
5
Cancer-related Molecular Pathways
5
Cellular Mechanics and Interactions
4
Papers citing this product
1
CaMKII locally encodes L-type channel activity to signal to nuclear CREB in excitation–transcription coupling
The Journal of Cell Biology
·
2008
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194 cit.
·
DOI
2
PTEN Regulates PI(3,4)P2 Signaling Downstream of Class I PI3K
Molecular Cell
·
2017
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182 cit.
·
DOI
3
Wnt-11 promotes neuroendocrine-like differentiation, survival and migration of prostate cancer cells
Molecular Cancer
·
2010
·
176 cit.
·
DOI
4
Induction of Cyclin D2 in Rat Granulosa Cells Requires FSH-dependent Relief from FOXO1 Repression Coupled with Positive Signals from Smad
Journal of Biological Chemistry
·
2004
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171 cit.
·
DOI
5
Glibenclamide Reduces Hippocampal Injury and Preserves Rapid Spatial Learning in a Model of Traumatic Brain Injury
Journal of Neuropathology & Experimental Neurology
·
2010
·
137 cit.
·
DOI
6
Differential expression of exosomal miRNAs between breast cancer patients with and without recurrence
Oncotarget
·
2017
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127 cit.
·
DOI
7
Curcumin Induces Autophagy, Apoptosis, and Cell Cycle Arrest in Human Pancreatic Cancer Cells
Evidence-based Complementary and Alternative Medicine
·
2017
·
111 cit.
·
DOI
8
Neuronal Atrophy Early in Degenerative Ataxia Is a Compensatory Mechanism to Regulate Membrane Excitability
Journal of Neuroscience
·
2015
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110 cit.
·
DOI
9
Androgen Receptor Remains Critical for Cell-Cycle Progression in Androgen-Independent CWR22 Prostate Cancer Cells
American Journal Of Pathology
·
2006
·
74 cit.
·
DOI
10
Loss of SMAD3 Promotes Vascular Remodeling in Pulmonary Arterial Hypertension via MRTF Disinhibition
American Journal of Respiratory and Critical Care Medicine
·
2017
·
68 cit.
·
DOI
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