Cell Signaling Technology

SimpleChIP® Human 28S rDNA Repeat Primers

#14901 · $137

68
Citing papers
2.1k
Combined citations
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Research areas

Mechanisms of cancer metastasis11Trace Elements in Health6Metal complexes synthesis and properties6Cell death mechanisms and regulation5Cancer Mechanisms and Therapy5Endoplasmic Reticulum Stress and Disease4NF-κB Signaling Pathways4Protein Kinase Regulation and GTPase Signaling4

Papers citing this product

1
Targeting iron homeostasis induces cellular differentiation and synergizes with differentiating agents in acute myeloid leukemia
The Journal of Experimental Medicine·2010·194 cit.·DOI
2
RACK1 Is an Insulin-like Growth Factor 1 (IGF-1) Receptor-interacting Protein That Can Regulate IGF-1-mediated Akt Activation and Protection from Cell Death
Journal of Biological Chemistry·2002·151 cit.·DOI
3
Angiotensin II impairs endothelial function via tyrosine phosphorylation of the endothelial nitric oxide synthase
The Journal of Experimental Medicine·2009·141 cit.·DOI
4
Cellular Iron Depletion Stimulates the JNK and p38 MAPK Signaling Transduction Pathways, Dissociation of ASK1-Thioredoxin, and Activation of ASK1
Journal of Biological Chemistry·2011·111 cit.·DOI
5
Dp44mT targets the AKT, TGF-β and ERK pathways via the metastasis suppressor NDRG1 in normal prostate epithelial cells and prostate cancer cells
British Journal of Cancer·2013·110 cit.·DOI
6
The Metastasis Suppressor, N-myc Downstream-regulated Gene 1 (NDRG1), Inhibits Stress-induced Autophagy in Cancer Cells
Journal of Biological Chemistry·2014·99 cit.·DOI
7
The metastasis suppressor, N-myc downstream regulated gene 1 (NDRG1), upregulates p21 via p53-independent mechanisms
Carcinogenesis·2011·91 cit.·DOI
8
The proto-oncogene c-Src and its downstream signaling pathways are inhibited by the metastasis suppressor, NDRG1
Oncotarget·2015·75 cit.·DOI
9
Zn<sup>2+</sup>Inhibits Mitochondrial Movement in Neurons by Phosphatidylinositol 3-Kinase Activation
Journal of Neuroscience·2005·75 cit.·DOI
10
Modulation of intracellular iron metabolism by iron chelation affects chromatin remodeling proteins and corresponding epigenetic modifications in breast cancer cells and increases their sensitivity to chemotherapeutic agents
International Journal of Oncology·2013·55 cit.·DOI