Cell Signaling Technology

MHC Class I (EMR8-5) Mouse Monoclonal Antibody (Alexa Fluor® 488 Conjugate)

#44238 · $465

17
Citing papers
428
Combined citations
Supplier page Find this product

Research areas

Mitochondrial Function and Pathology7Ion channel regulation and function4MicroRNA in disease regulation3Metabolomics and Mass Spectrometry Studies3Cancer, Hypoxia, and Metabolism2Neuroscience and Neuropharmacology Research2Metabolism, Diabetes, and Cancer2NF-κB Signaling Pathways2

Papers citing this product

1
CCC- and WASH-mediated endosomal sorting of LDLR is required for normal clearance of circulating LDL
Nature Communications·2016·229 cit.·DOI
2
Insulin-induced β-Arrestin1 Ser-412 Phosphorylation Is a Mechanism for Desensitization of ERK Activation by Gαi-coupled Receptors
Journal of Biological Chemistry·2004·43 cit.·DOI
3
A Ca<sup>2+</sup>-Dependent Mechanism Boosting Glycolysis and OXPHOS by Activating Aralar-Malate-Aspartate Shuttle, upon Neuronal Stimulation
Journal of Neuroscience·2022·39 cit.·DOI
4
miR-622 is a novel potential biomarker of breast carcinoma and impairs motility of breast cancer cells through targeting NUAK1 kinase
British Journal of Cancer·2020·37 cit.·DOI
5
The Low-Density Lipoprotein Receptor Is Regulated by Estrogen and Forms a Functional Complex with the Estrogen-Regulated Protein Ezrin in Pituitary GH3 Somatolactotropes
Endocrinology·2004·26 cit.·DOI
6
IKCa channels control breast cancer metabolism including AMPK-driven autophagy
Cell Death and Disease·2022·24 cit.·DOI
7
EFLDO sensitizes liver cancer cells to TNFSF10‑induced apoptosis in a p53‑dependent manner
Molecular Medicine Reports·2019·11 cit.·DOI
8
ATM is activated by ATP depletion and modulates mitochondrial function through NRF1
bioRxiv (Cold Spring Harbor Laboratory)·2018·6 cit.·DOI
9
miR-622 Counteracts the NUAK1-Induced Gastric Cancer Cell Proliferation and the Antioxidative Stress
Disease Markers·2022·5 cit.·DOI
10
Real-time resolution studies of the regulation of pyruvate-dependent lactate metabolism by hexokinases in single cells
PLoS ONE·2023·4 cit.·DOI

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