Cell Signaling Technology

Jagged1 (D4Y1R) Rabbit Monoclonal Antibody

#70109 · $178

40
Citing papers
715
Combined citations
Supplier page Find this product

Research areas

Cancer, Hypoxia, and Metabolism6Cancer-related molecular mechanisms research6MicroRNA in disease regulation4Cancer Cells and Metastasis3Cancer Genomics and Diagnostics3Epigenetics and DNA Methylation3Cancer Immunotherapy and Biomarkers2Adipose Tissue and Metabolism2

Papers citing this product

1
ASPH-notch Axis guided Exosomal delivery of Prometastatic Secretome renders breast Cancer multi-organ metastasis
Molecular Cancer·2019·120 cit.·DOI
2
Lactate-activated macrophages induced aerobic glycolysis and epithelial-mesenchymal transition in breast cancer by regulation of CCL5-CCR5 axis: a positive metabolic feedback loop
Oncotarget·2017·91 cit.·DOI
3
Novel Histopathological Patterns in Cortical Tubers of Epilepsy Surgery Patients with Tuberous Sclerosis Complex
PLoS ONE·2016·89 cit.·DOI
4
Lutein inhibits proliferation, invasion and migration of hypoxic breast cancer cells via downregulation of HES1
International Journal of Oncology·2018·70 cit.·DOI
5
Hypertension reduces soluble guanylyl cyclase expression in the mouse aorta via the Notch signaling pathway
Scientific Reports·2017·46 cit.·DOI
6
Increased hemoglobin and heme in MALDI-TOF MS analysis induce ferroptosis and promote degeneration of herniated human nucleus pulposus
Molecular Medicine·2021·44 cit.·DOI
7
JAGGED1/NOTCH3 activation promotes aortic hypermuscularization and stenosis in elastin deficiency
Journal of Clinical Investigation·2022·35 cit.·DOI
8
Screening of compounds to identify novel epigenetic regulatory factors that affect innate immune memory in macrophages
Scientific Reports·2022·27 cit.·DOI
9
Long Non-coding RNA KCNQ1OT1 Contributes to Antiepileptic Drug Resistance Through the miR-138-5p/ABCB1 Axis in vitro
Frontiers in Neuroscience·2019·25 cit.·DOI
10
Association of Jagged1 expression with malignancy and prognosis in human pancreatic cancer
Cellular Oncology·2020·24 cit.·DOI

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