Cell Signaling Technology

Sin1 (D7G1A) Rabbit Monoclonal Antibody

#12860 · $161

80
Citing papers
1.9k
Combined citations
Supplier page Find this product

Research areas

Autoimmune Bullous Skin Diseases8Coagulation, Bradykinin, Polyphosphates, and Angioedema7Heat shock proteins research5Urticaria and Related Conditions4Autophagy in Disease and Therapy4Angiogenesis and VEGF in Cancer4Genetics and Neurodevelopmental Disorders4Cancer-related Molecular Pathways3

Papers citing this product

1
IL-6- and NGF-Induced Rapid Control of Protein Synthesis and Nociceptive Plasticity via Convergent Signaling to the eIF4F Complex
Journal of Neuroscience·2010·225 cit.·DOI
2
Chlamydial Entry Involves TARP Binding of Guanine Nucleotide Exchange Factors
PLoS Pathogens·2008·153 cit.·DOI
3
Cryptotanshinone from the Salvia miltiorrhiza Bunge Attenuates Ethanol-Induced Liver Injury by Activation of AMPK/SIRT1 and Nrf2 Signaling Pathways
International Journal of Molecular Sciences·2019·142 cit.·DOI
4
MiR-503 modulates epithelial-mesenchymal transition in silica-induced pulmonary fibrosis by targeting PI3K p85 and is sponged by lncRNA MALAT1
Scientific Reports·2017·110 cit.·DOI
5
Autoantibodies in the Autoimmune Disease Pemphigus Foliaceus Induce Blistering via p38 Mitogen-Activated Protein Kinase-Dependent Signaling in the Skin
American Journal Of Pathology·2008·94 cit.·DOI
6
p38 MAPK Activation Is Downstream of the Loss of Intercellular Adhesion in Pemphigus Vulgaris
Journal of Biological Chemistry·2010·84 cit.·DOI
7
TMBIM6/BI-1 contributes to cancer progression through assembly with mTORC2 and AKT activation
Nature Communications·2020·80 cit.·DOI
8
A Pathophysiologic Role for Epidermal Growth Factor Receptor in Pemphigus Acantholysis
Journal of Biological Chemistry·2013·66 cit.·DOI
9
Amino acid deprivation induces AKT activation by inducing GCN2/ATF4/REDD1 axis
Cell Death and Disease·2021·59 cit.·DOI
10
PERK recruits E-Syt1 at ER–mitochondria contacts for mitochondrial lipid transport and respiration
The Journal of Cell Biology·2023·57 cit.·DOI