R&D Systems

Mouse IgG Horseradish Peroxidase-conjugated Antibody

#HAF007 · western blot

200
Citing papers
4k
Combined citations
Supplier page Find this product

Research areas

Cancer Cells and Metastasis5Autophagy in Disease and Therapy4Cell death mechanisms and regulation4Endoplasmic Reticulum Stress and Disease4Neuroinflammation and Neurodegeneration Mechanisms4Cancer, Lipids, and Metabolism3Blood Coagulation and Thrombosis Mechanisms3Cell Adhesion Molecules Research3

Papers citing this product

1
Global analysis of genome, transcriptome and proteome reveals the response to aneuploidy in human cells
Molecular Systems Biology·2012·485 cit.·DOI
2
The fungal peptide toxin Candidalysin activates the NLRP3 inflammasome and causes cytolysis in mononuclear phagocytes
Nature Communications·2018·283 cit.·DOI
3
HIV and HCV Activate the Inflammasome in Monocytes and Macrophages via Endosomal Toll-Like Receptors without Induction of Type 1 Interferon
PLoS Pathogens·2014·177 cit.·DOI
4
MicroRNA-7 Regulates the Function of Mitochondrial Permeability Transition Pore by Targeting VDAC1 Expression
Journal of Biological Chemistry·2016·130 cit.·DOI
5
Induction of 3-hydroxy-3-methylglutaryl-CoA reductase mediates statin resistance in breast cancer cells
Cell Death and Disease·2019·118 cit.·DOI
6
MicroRNA-7 Protects against 1-Methyl-4-Phenylpyridinium-Induced Cell Death by Targeting RelA
Journal of Neuroscience·2014·104 cit.·DOI
7
Lung-resident memory B cells protect against bacterial pneumonia
Journal of Clinical Investigation·2021·104 cit.·DOI
8
Angiotensin II triggers release of neutrophil extracellular traps, linking thromboinflammation with essential hypertension
JCI Insight·2021·94 cit.·DOI
9
Epigallocatechin-3-gallate (EGCG) Alters Histone Acetylation and Methylation and Impacts Chromatin Architecture Profile in Human Endothelial Cells
Molecules·2020·76 cit.·DOI
10
Lutein inhibits proliferation, invasion and migration of hypoxic breast cancer cells via downregulation of HES1
International Journal of Oncology·2018·70 cit.·DOI

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