Cell Signaling Technology

Fer (5D2) Mouse Monoclonal Antibody

#4268 · $158,348

60

Evidence Score · Well-Established

Used by 92 independent labs across 100 papers. How we score this →

100

Citing papers

7.6k

Combined citations

Supplier page Find this product

Research areas

PI3K/AKT/mTOR signaling in cancer11 Cell death mechanisms and regulation7 Ubiquitin and proteasome pathways7 Cancer-related Molecular Pathways6 Pancreatic function and diabetes6 Lung Cancer Treatments and Mutations6 Protein Degradation and Inhibitors6 Cancer-related molecular mechanisms research5

Papers citing this product

1

HDAC1 modulates OGG1-initiated oxidative DNA damage repair in the aging brain and Alzheimer’s disease

Nature Communications·2020·209 cit.·DOI

2

Enhancing intracellular accumulation and target engagement of PROTACs with reversible covalent chemistry

Nature Communications·2020·204 cit.·DOI

3

Multiple ABCB1 transcriptional fusions in drug resistant high-grade serous ovarian and breast cancer

Nature Communications·2019·195 cit.·DOI

4

Conformational switching of the pseudokinase domain promotes human MLKL tetramerization and cell death by necroptosis

Nature Communications·2018·192 cit.·DOI

5

PI3K/AKT/mTOR and sonic hedgehog pathways cooperate together to inhibit human pancreatic cancer stem cell characteristics and tumor growth

Oncotarget·2015·160 cit.·DOI

6

HSP90 activity is required for MLKL oligomerisation and membrane translocation and the induction of necroptotic cell death

Cell Death and Disease·2016·158 cit.·DOI

7

Effects of Intestinal Microbiota on Brain Development in Humanized Gnotobiotic Mice

Scientific Reports·2018·149 cit.·DOI

8

RIP1 inhibition blocks inflammatory diseases but not tumor growth or metastases

Cell Death and Differentiation·2019·145 cit.·DOI

9

<i>db</i>/<i>db</i>Mice Exhibit Features of Human Type 2 Diabetes That Are Not Present in Weight-Matched C57BL/6J Mice Fed a Western Diet

Journal of Diabetes Research·2017·145 cit.·DOI

10

Optimal B-cell proliferation requires phosphoinositide 3-kinase–dependent inactivation of FOXO transcription factors

Blood·2004·143 cit.·DOI

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