Cell Signaling Technology

Immunofluorescence Application Solutions Kit

#12727 · immunofluorescence · $149

100
Citing papers
3.8k
Combined citations
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Research areas

Epigenetics and DNA Methylation11MicroRNA in disease regulation8Cancer-related gene regulation7Cancer, Hypoxia, and Metabolism6Cancer-related molecular mechanisms research5Pregnancy and preeclampsia studies5Protein Degradation and Inhibitors5Immune cells in cancer4

Papers citing this product

1
High-efficiency optogenetic silencing with soma-targeted anion-conducting channelrhodopsins
Nature Communications·2018·331 cit.·DOI
2
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation
Journal of Experimental & Clinical Cancer Research·2018·230 cit.·DOI
3
Cell-cycle arrest and senescence in TP53-wild type renal carcinoma by enhancer RNA-P53-bound enhancer regions 2 (p53BER2) in a p53-dependent pathway
Cell Death and Disease·2021·157 cit.·DOI
4
<i>Cordyceps cicadae</i> Mycelia Ameliorate Cisplatin-Induced Acute Kidney Injury by Suppressing the TLR4/NF-<i>κ</i>B/MAPK and Activating the HO-1/Nrf2 and Sirt-1/AMPK Pathways in Mice
Oxidative Medicine and Cellular Longevity·2020·127 cit.·DOI
5
ANXA4 promotes trophoblast invasion via the PI3K/Akt/eNOS pathway in preeclampsia
American Journal of Physiology-Cell Physiology·2019·126 cit.·DOI
6
The lncRNA TUG1 modulates proliferation in trophoblast cells via epigenetic suppression of RND3
Cell Death and Disease·2017·121 cit.·DOI
7
Regulation of Rod Phototransduction Machinery by Ciliary Neurotrophic Factor
Journal of Neuroscience·2006·90 cit.·DOI
8
EZH2-mediated epigenetic silencing of TIMP2 promotes ovarian cancer migration and invasion
Scientific Reports·2017·88 cit.·DOI
9
Entire mitogen activated protein kinase (MAPK) pathway is present in preimplantation mouse embryos
Developmental Dynamics·2004·77 cit.·DOI
10
Exosomes from Plasmodium-infected hosts inhibit tumor angiogenesis in a murine Lewis lung cancer model
Oncogenesis·2017·71 cit.·DOI

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