Cell Signaling Technology

USP8 (D18F6) Rabbit Monoclonal Antibody

#11832 · $161

84
Citing papers
2.2k
Combined citations
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Research areas

Bioactive Compounds and Antitumor Agents13Cancer therapeutics and mechanisms8Cancer-related gene regulation6Alzheimer's disease research and treatments5Neuroscience and Neuropharmacology Research5T-cell and Retrovirus Studies5Blood Coagulation and Thrombosis Mechanisms4RNA modifications and cancer4

Papers citing this product

1
Loss of α7 Nicotinic Receptors Enhances β-Amyloid Oligomer Accumulation, Exacerbating Early-Stage Cognitive Decline and Septohippocampal Pathology in a Mouse Model of Alzheimer's Disease
Journal of Neuroscience·2010·206 cit.·DOI
2
β-Lapachone induces programmed necrosis through the RIP1-PARP-AIF-dependent pathway in human hepatocellular carcinoma SK-Hep1 cells
Cell Death and Disease·2014·103 cit.·DOI
3
Anti–neurofascin-155 IgG4 antibodies prevent paranodal complex formation in vivo
Journal of Clinical Investigation·2019·103 cit.·DOI
4
Differential Roles of Insulin Receptor Substrates in the Anti-apoptotic Function of Insulin-like Growth Factor-1 and Insulin
Journal of Biological Chemistry·2002·97 cit.·DOI
5
USP10 modulates the SKP2/Bcr-Abl axis via stabilizing SKP2 in chronic myeloid leukemia
Cell Discovery·2019·97 cit.·DOI
6
An NQO1-Initiated and p53-Independent Apoptotic Pathway Determines the Anti-Tumor Effect of Tanshinone IIA against Non-Small Cell Lung Cancer
PLoS ONE·2012·91 cit.·DOI
7
Galectin-9 Protein Is Up-regulated in Astrocytes by Tumor Necrosis Factor and Promotes Encephalitogenic T-cell Apoptosis
Journal of Biological Chemistry·2013·86 cit.·DOI
8
Vitamin C Restricts the Emergence of Acquired Resistance to EGFR-Targeted Therapies in Colorectal Cancer
Cancers·2020·72 cit.·DOI
9
Roles of 14-3-3 and calmodulin binding in subcellular localization and function of the small G-protein Rem2
Biochemical Journal·2005·65 cit.·DOI
10
The HSP70 co-chaperone DNAJC14 targets misfolded pendrin for unconventional protein secretion
Nature Communications·2016·63 cit.·DOI

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