Agilent/Dako

S100A4 (MTS1)

#A5114 · immunohistochemistry

63
Evidence Score · Well-Established
Used by 117 independent labs across 144 papers. How we score this →
43
Citing papers
3.9k
Combined citations
Supplier page Find this product

Research areas

S100 Proteins and Annexins8Chronic Kidney Disease and Diabetes6Renal and related cancers5Neonatal Respiratory Health Research4Chronic Obstructive Pulmonary Disease (COPD) Research4Congenital heart defects research4Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis3Dialysis and Renal Disease Management3

Papers citing this product

1
Fate Tracing Reveals the Pericyte and Not Epithelial Origin of Myofibroblasts in Kidney Fibrosis
American Journal Of Pathology·2009·1.4k cit.·DOI
2
Tubular Overexpression of Transforming Growth Factor-β1 Induces Autophagy and Fibrosis but Not Mesenchymal Transition of Renal Epithelial Cells
American Journal Of Pathology·2010·271 cit.·DOI
3
Single-Cell RNA Sequencing Reveals Renal Endothelium Heterogeneity and Metabolic Adaptation to Water Deprivation
Journal of the American Society of Nephrology·2019·194 cit.·DOI
4
Vitamin D receptor expression and associated gene signature in tumour stromal fibroblasts predict clinical outcome in colorectal cancer
Gut·2016·188 cit.·DOI
5
Klotho Ameliorates Kidney Injury and Fibrosis and Normalizes Blood Pressure by Targeting the Renin-Angiotensin System
American Journal Of Pathology·2015·160 cit.·DOI
6
Genetic lineage tracing identifies in situ Kit-expressing cardiomyocytes
Cell Research·2015·141 cit.·DOI
7
Phenotype of airway epithelial cells suggests epithelial to mesenchymal cell transition in clinically stable lung transplant recipients
Thorax·2005·125 cit.·DOI
8
c-Met–mediated endothelial plasticity drives aberrant vascularization and chemoresistance in glioblastoma
Journal of Clinical Investigation·2016·123 cit.·DOI
9
Expression of S100A4 by a variety of cell types present in the tumor microenvironment of human breast cancer
International Journal of Cancer·2007·120 cit.·DOI
10
MyD88 Signaling Pathway Is Involved in Renal Fibrosis by Favoring a TH2 Immune Response and Activating Alternative M2 Macrophages
Molecular Medicine·2012·111 cit.·DOI

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