Cell Signaling Technology

nNOS (C7D7) Rabbit Monoclonal Antibody

#4231 · $156

100
Citing papers
10.3k
Combined citations
Supplier page Find this product

Research areas

Alzheimer's disease research and treatments6Neuroinflammation and Neurodegeneration Mechanisms6Pluripotent Stem Cells Research5MicroRNA in disease regulation5Epigenetics and DNA Methylation5Neuroscience and Neuropharmacology Research5Hippo pathway signaling and YAP/TAZ5Cancer-related molecular mechanisms research4

Papers citing this product

1
β-Amyloid Monomers Are Neuroprotective
Journal of Neuroscience·2009·406 cit.·DOI
2
B cells sustain inflammation and predict response to immune checkpoint blockade in human melanoma
Nature Communications·2019·374 cit.·DOI
3
Activation of Placental mTOR Signaling and Amino Acid Transporters in Obese Women Giving Birth to Large Babies
The Journal of Clinical Endocrinology & Metabolism·2012·303 cit.·DOI
4
S100B and S100A6 Differentially Modulate Cell Survival by Interacting with Distinct RAGE (Receptor for Advanced Glycation End Products) Immunoglobulin Domains
Journal of Biological Chemistry·2007·262 cit.·DOI
5
Cancer-associated fibroblasts enhance metastatic potential of lung cancer cells through IL-6/STAT3 signaling pathway
Oncotarget·2017·221 cit.·DOI
6
Dissecting the dynamics of signaling events in the BMP, WNT, and NODAL cascade during self-organized fate patterning in human gastruloids
PLoS Biology·2019·209 cit.·DOI
7
Mechanisms of Locomotor Sensitization to Drugs of Abuse in a Two-Injection Protocol
Neuropsychopharmacology·2009·205 cit.·DOI
8
Functional Crosstalk between Type I and II Interferon through the Regulated Expression of STAT1
PLoS Biology·2010·196 cit.·DOI
9
BCL2/BCL-XL inhibition induces apoptosis, disrupts cellular calcium homeostasis, and prevents platelet activation
Blood·2011·189 cit.·DOI
10
Memantine Rescues Transient Cognitive Impairment Caused by High-Molecular-Weight A  Oligomers But Not the Persistent Impairment Induced by Low-Molecular-Weight Oligomers
Journal of Neuroscience·2013·185 cit.·DOI

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