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Millipore
Anti-Histone H3.3 Antibody, K27M mutant
#ABE419 · western blot
39
Citing papers
2.3k
Combined citations
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Research areas
Glioma Diagnosis and Treatment
35
Chromatin Remodeling and Cancer
11
Neurofibromatosis and Schwannoma Cases
10
Epigenetics and DNA Methylation
7
Cancer Genomics and Diagnostics
5
Meningioma and schwannoma management
4
Brain Metastases and Treatment
3
MicroRNA in disease regulation
3
Papers citing this product
1
H3K27M induces defective chromatin spread of PRC2-mediated repressive H3K27me2/me3 and is essential for glioma tumorigenesis
Nature Communications
·
2019
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384 cit.
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DOI
2
Spatial and temporal homogeneity of driver mutations in diffuse intrinsic pontine glioma
Nature Communications
·
2016
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262 cit.
·
DOI
3
Novel and shared neoantigen derived from histone 3 variant H3.3K27M mutation for glioma T cell therapy
The Journal of Experimental Medicine
·
2017
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215 cit.
·
DOI
4
Detection of Histone H3 mutations in cerebrospinal fluid-derived tumor DNA from children with diffuse midline glioma
Acta Neuropathologica Communications
·
2017
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172 cit.
·
DOI
5
A sensitive and specific histopathologic prognostic marker for H3F3A K27M mutant pediatric glioblastomas
Acta Neuropathologica
·
2014
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147 cit.
·
DOI
6
Specific detection of methionine 27 mutation in histone 3 variants (H3K27M) in fixed tissue from high-grade astrocytomas
Acta Neuropathologica
·
2014
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137 cit.
·
DOI
7
Incidence and clinicopathologic features of H3 K27M mutations in adults with radiographically-determined midline gliomas
Journal of Neuro-Oncology
·
2019
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127 cit.
·
DOI
8
The molecular characteristics of spinal cord gliomas with or without H3 K27M mutation
Acta Neuropathologica Communications
·
2020
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120 cit.
·
DOI
9
Comprehensive molecular characterisation of epilepsy-associated glioneuronal tumours
Acta Neuropathologica
·
2017
·
75 cit.
·
DOI
10
BRAF Fusion Analysis in Pilocytic Astrocytomas: KIAA1549-BRAF 15-9 Fusions Are More Frequent in the Midline Than Within the Cerebellum
Journal of Neuropathology & Experimental Neurology
·
2015
·
74 cit.
·
DOI
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