Cell Signaling Technology

Smac/Diablo (79-1-83) Mouse Monoclonal Antibody

#2954 · $158

100
Citing papers
16.3k
Combined citations
Supplier page Find this product

Research areas

PI3K/AKT/mTOR signaling in cancer12MicroRNA in disease regulation7Neuroinflammation and Neurodegeneration Mechanisms5Immune Cell Function and Interaction5Cancer-related Molecular Pathways5Epigenetics and DNA Methylation5Antifungal resistance and susceptibility5Cytokine Signaling Pathways and Interactions5

Papers citing this product

1
Candidalysin is a fungal peptide toxin critical for mucosal infection
Nature·2016·891 cit.·DOI
2
Research Resource: Comprehensive Expression Atlas of the Fibroblast Growth Factor System in Adult Mouse
Molecular Endocrinology·2010·679 cit.·DOI
3
An amino-bisphosphonate targets MMP-9–expressing macrophages and angiogenesis to impair cervical carcinogenesis
Journal of Clinical Investigation·2004·609 cit.·DOI
4
An amino-bisphosphonate targets MMP-9–expressing macrophages and angiogenesis to impair cervical carcinogenesis
Journal of Clinical Investigation·2004·576 cit.·DOI
5
Response of a Neuronal Model of Tuberous Sclerosis to Mammalian Target of Rapamycin (mTOR) Inhibitors: Effects on mTORC1 and Akt Signaling Lead to Improved Survival and Function
Journal of Neuroscience·2008·502 cit.·DOI
6
PTEN posttranslational inactivation and hyperactivation of the PI3K/Akt pathway sustain primary T cell leukemia viability
Journal of Clinical Investigation·2008·441 cit.·DOI
7
Interplay of IKK/NF-κB signaling in macrophages and myofibers promotes muscle degeneration in Duchenne muscular dystrophy
Journal of Clinical Investigation·2007·432 cit.·DOI
8
Elevated Phosphatidylinositol 3,4,5-Trisphosphate in Glia Triggers Cell-Autonomous Membrane Wrapping and Myelination
Journal of Neuroscience·2010·349 cit.·DOI
9
The oncogenic properties of mutant p110α and p110β phosphatidylinositol 3-kinases in human mammary epithelial cells
Proceedings of the National Academy of Sciences·2005·327 cit.·DOI
10
RETRACTED: Breast Tumor Cells with PI3K Mutation or HER2 Amplification Are Selectively Addicted to Akt Signaling
PLoS ONE·2008·317 cit.·DOI

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